Faculty

Institution

Unit

Venue

Xiao-Mei Qi, MD

Associate Professor

Medical College of Wisconsin

Affiliations

Departments of Pharmacology and Toxicology

Xiao-mei Qi, Md is currently serving as a Departments Of Pharmacology And Toxicology Associate Professor at Medical College of Wisconsin.

Honors

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Experiences

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Education

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Xiao-Mei Qi, MD

Medical College of Wisconsin

Associate Professor

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cell signaling molecular biology cell biology signal transduction biochemistry oncogenes signaling pathways tumor biology biological chemistry cancer biology molecular cell biology cancer research systems biology protein phosphorylation gene expression breast cancer cancer cells cell regulation estrogen receptor oncology therapeutic targets protein science breast oncology aerobic glycolysis pancreatic tumorigenesis medicinal research molecular physiology cell death apoptosis transcription regulation biochemical sciences clinical biochemistry cellular physiology enzyme inhibition cancer discovery cancer treatment protein function inhibitory activity metalloproteins colorectal cancer cancer genetics stress response opposite effects jnk pathways ras transformation c-jun-dependent ubiquitin-proteasome pathways breast cancer invasion c-jun transcription cancer hormone sensitivity mammalian biology vascular biology cell metabolism biochemical genetics functional selectivity anti-cancer agents pancreatic cancer mitophagy primary metabolite cancer genomics pathway analysis host factors cytokines biological inorganic chemistry cell control cancer immunosurveillance tumor targeting inflammatory diseases metabolic regulation metabolic networks insulin signaling protein expression k-ras transformation triple-negative breast cancer c-jun activity er phosphorylation nonclassical pathway molecular cancer pharmacology cancer letter hormonal receptor

Notable Scholarly Works

Notable Federal Grants

Collaborators

CORRECTION: P38γ Mapk is Essential for Aerobic Glycolysis and Pancreatic Tumorigenesis. Cancer Research (2025)

Fang WangXiao-Mei QiRyan WertzMatthew MortensenCatherine HagenJohn EvansYuri SheininMichael JamesPengyuan LiuSusan TsaiJames ThomasAlexander MackinnonMichael DwinellCharles R. MyersRamon Bartrons BachLiwu FuGuan Chen

tumor biologyoncologycancer researchbiomolecular engineeringcancer biologypancreatic tumorigenesisaerobic glycolysismolecular biology

Protein Tyrosine Phosphatase Ptph1 Potentiates Receptor Tyrosine Kinase Her2 Oncogenesis via a Pdz-Coupled and Phosphorylation-Driven Scaffold. American Journal of Cancer Research (2024)

Xiaomei QiFang WangLinda ThomasShao MaKatie PalenYan LuYuri SheininJill GershanLiwu FuGuan Chen

receptorstumor biologyprotein phosphorylationoncologycancer researchcancer biologyoncogenesbiochemistryphosphorylation-driven scaffoldreceptor tyrosine kinasesmolecular biology

Data From Ptph1 Dephosphorylates and Cooperates With P38γ Mapk to Increase Ras Oncogenesis Through Pdz-Mediated Interaction. (2023)

Song-Wang HouHui-Ying ZhiNicole PohlMathew LoeschXiao-Mei QiRong-Shan LiZainab BasirGuan Chen

molecular biologyprotein phosphorylationpdz-mediated interactionras oncogenesis

Figure S6 From P38γ Mapk is Essential for Aerobic Glycolysis and Pancreatic Tumorigenesis. (2023)

pancreatic tumorigenesisbiochemistrytumor biologyaerobic glycolysis

P38γ Mapk Inflammatory and Metabolic Signaling in Physiology and Disease. Cells (2023)

Xiao-Mei QiGuan Chen

metabolic pathwaysimmune responsessignaling pathwaysinsulin signalinginflammationmetabolic signalingcell biologycell signalingmetabolic networksinflammatory diseasessystems biologyphysiologysignal transductionmetabolic regulationcellular physiologymolecular physiology

Supplementary Data From P38γ Mapk is Essential for Aerobic Glycolysis and Pancreatic Tumorigenesis. (2023)

pancreatic tumorigenesisbiochemistryaerobic glycolysis

Isoform-Specific and Cell/Tissue-Dependent Effects of P38 Mapks in Regulating Inflammation and Inflammation-Associated Oncogenesis. Frontiers in Bioscience-Landmark (2022)

Jian-Zhong QinHong XinXiao-Mei QiGuan Chen

inflammation-associated oncogenesisinflammationcancer biologycell biologycell signalingsystems biologymolecular biology

P38γ Mapk is Essential for Aerobic Glycolysis and Pancreatic Tumorigenesis. Cancer Research (2020)

primary metabolitecancer biologytumor biologyoncologycancer researchpancreatic tumorigenesismitophagysystems biologyglycobiologycell metabolismtumor microenvironmentprotein phosphorylationpancreatic cancerbiochemistryvascular biologyaerobic glycolysiscancer cellsoncogenesmolecular biologycellular physiology

Nogo-B Receptor Promotes Epithelial-Mesenchymal Transition in Non-Small Cell Lung Cancer Cells Through the Ras/Erk/Snail1 Pathway. Cancer Lett (2018)

metastasislung cancertumor biologynogo-b receptorcancer growthradiation oncologycancer biologyras/erk/snail1 pathwaycell biologycell signalingcancer cellsoncogenesthoracic oncologycarcinogenesiscancer geneticsepithelial-mesenchymal transition

Targeting an Oncogenic Kinase/Phosphatase Signaling Network for Cancer Therapy. Acta Pharmaceutica Sinica B (2018)

Xiao-Mei QiFang WangMatthew MortensenRyan WertzGuan Chen

signaling pathwayscancer researchoncologycancer biologycancer therapyoncogenespharmacologycancer treatmenttherapeutic targetstumor targeting

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Xiao-Mei Qi, MD

Associate Professor

Medical College of Wisconsin

Affiliations

Departments of Pharmacology and Toxicology

Xiao-mei Qi, Md is currently serving as a Departments Of Pharmacology And Toxicology Associate Professor at Medical College of Wisconsin.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Xiao-Mei Qi, MD

Medical College of Wisconsin

Associate Professor

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

CORRECTION: P38γ Mapk is Essential for Aerobic Glycolysis and Pancreatic Tumorigenesis. Cancer Research (2025)

tumor biologyoncologycancer researchbiomolecular engineeringcancer biologypancreatic tumorigenesisaerobic glycolysismolecular biology

Protein Tyrosine Phosphatase Ptph1 Potentiates Receptor Tyrosine Kinase Her2 Oncogenesis via a Pdz-Coupled and Phosphorylation-Driven Scaffold. American Journal of Cancer Research (2024)

Xiaomei QiFang WangLinda ThomasShao MaKatie PalenYan LuYuri SheininJill GershanLiwu FuGuan Chen

receptorstumor biologyprotein phosphorylationoncologycancer researchcancer biologyoncogenesbiochemistryphosphorylation-driven scaffoldreceptor tyrosine kinasesmolecular biology

Data From Ptph1 Dephosphorylates and Cooperates With P38γ Mapk to Increase Ras Oncogenesis Through Pdz-Mediated Interaction. (2023)

Song-Wang HouHui-Ying ZhiNicole PohlMathew LoeschXiao-Mei QiRong-Shan LiZainab BasirGuan Chen

molecular biologyprotein phosphorylationpdz-mediated interactionras oncogenesis

Figure S6 From P38γ Mapk is Essential for Aerobic Glycolysis and Pancreatic Tumorigenesis. (2023)

pancreatic tumorigenesisbiochemistrytumor biologyaerobic glycolysis

P38γ Mapk Inflammatory and Metabolic Signaling in Physiology and Disease. Cells (2023)

Xiao-Mei QiGuan Chen

Supplementary Data From P38γ Mapk is Essential for Aerobic Glycolysis and Pancreatic Tumorigenesis. (2023)

pancreatic tumorigenesisbiochemistryaerobic glycolysis

Isoform-Specific and Cell/Tissue-Dependent Effects of P38 Mapks in Regulating Inflammation and Inflammation-Associated Oncogenesis. Frontiers in Bioscience-Landmark (2022)

Jian-Zhong QinHong XinXiao-Mei QiGuan Chen

inflammation-associated oncogenesisinflammationcancer biologycell biologycell signalingsystems biologymolecular biology

P38γ Mapk is Essential for Aerobic Glycolysis and Pancreatic Tumorigenesis. Cancer Research (2020)

Nogo-B Receptor Promotes Epithelial-Mesenchymal Transition in Non-Small Cell Lung Cancer Cells Through the Ras/Erk/Snail1 Pathway. Cancer Lett (2018)

Targeting an Oncogenic Kinase/Phosphatase Signaling Network for Cancer Therapy. Acta Pharmaceutica Sinica B (2018)

Xiao-Mei QiFang WangMatthew MortensenRyan WertzGuan Chen

signaling pathwayscancer researchoncologycancer biologycancer therapyoncogenespharmacologycancer treatmenttherapeutic targetstumor targeting

1
2
3
4
10 / page

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Similar Faculty Members

Pau Castel

Institution

New York University

oncogene

tumor biology

Brian Rowan

Institution

Tulane University

cancer letter

health science

Cheng-Ming Chiang

Institution

University of Texas Southwestern Medical Center

cancer letter

chromatin remodeling

John Blenis

Institution

Weill Cornell Medicine

molecular biology

cell signaling

Lee Graves

Institution

University of North Carolina at Chapel Hill

molecular biology

pharmacology

Steffi Oesterreich

Institution

University of Pittsburgh

cancer letter

pathology

Michael A Mancini

Institution

Baylor College of Medicine

molecular cell biology

cell biology

Danny Dhanasekaran

Institution

University of Oklahoma

metabolomic variations

transcriptome

Jamboor Vishwanatha

Institution

University of North Texas Health Science Center

neprilysin

neoplasm metastasis

Cress, Douglas

Institution

University of South Florida

cell biology

oncogenes