Faculty

Institution

Unit

Venue

Suresh Palaniyandi

Assistant Professor

Wayne State University

Affiliations

Graduate School

School of Medicine

Physiology

Suresh Palaniyandi is currently serving as a Graduate School Assistant Professor at Wayne State University.

Honors

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Experiences

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Education

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Suresh Palaniyandi

Wayne State University

Assistant Professor

You can click a keyword to filter the displayed items

pharmacology cardiology physiology cardiovascular research biochemistry membrane biology diabetes heart failure mammalian biology aldehyde dehydrogenase inflammation medicine medicinal research cellular physiology vascular biology circulatory physiology redox biology liver physiology cardiomyopathy cell signaling molecular medicine endocrinology enzyme inhibition molecular physiology free radical biology mitochondrial function cellular biochemistry protein science angiogenesis endothelial dysfunction experimental medicine biological function angiology diabetic cardiomyopathy protein function cardiovascular pharmacology signaling pathways pathway analysis cardiovascular function pharmacological research experimental gerontology adaptive immune system experimental biology cell physiology redox signaling cellular senescence biogerontology cell regulation comparative effects senescence-accelerated prone mice mast cells metabolic regulation insulin signaling pharmaceutical sciences pharmaceutical research experimental pharmacology experimental toxicology tissue physiology innate immune system short-term variability fibrosis neuroprotection knockout mice cardiac remodeling endoplasmic reticulum stress beneficial effects coronary endothelial cells biomedical modeling heart disease therapeutic strategies macrophage polarization streptozotocin-induced diabetic mice olmesartan treatment high-fat diet cardiomyocyte hypertrophy low-dose streptozotocin

Notable Scholarly Works

Notable Federal Grants

Collaborators

Aldh2 and NfκB Activation Restores Vegfr2 Expression and Angiogenesis Impaired by 4‐Hne in Coronary Endothelial Cells. Cell Biochemistry and Function (2026)

inflammationcardiovascular researchfree radical biologyneovascularizationcell signalingblood vesselsvascular biologycell biologyangiogenesisendothelial dysfunctionphysiology

Loss of Hepatic Aldh2 Contributes to Metabolic Stress Mediated Heart Failure. Physiology (2025)

health sciencescardiovascular researchliver physiologymetabolic syndromestress (biology)circulatory physiologydisease pathophysiologyheart failuremetabolismphysiology

Maresin-1 Promotes Neuroprotection and Modulates Metabolic and Inflammatory Responses in Disease-Associated Cell Types in Preclinical Models of Multiple Sclerosis. Journal of Biological Chemistry (2025)

neurological progressmultiple sclerosisinflammationmolecular neurodegenerationneuroprotectioncell biologyneuroinflammation

Targeted Overexpression of Mitochondrial Aldh2 in Coronary Endothelial Cells Mitigates Hfpef in a Diabetic Mouse Model. Biomolecules (2025)

mitochondrial functioncardiovascular researchcell metabolismdiabetes researchcell signalingvascular biologycell biologyendothelial dysfunctionphysiology

Therapeutic Strategies for Reversing Cardiac Fibrosis in Hearts Supported by Ventricular Assist Devices. Drug Discovery Today (2025)

pharmacologymedicinedrug discoverydevice therapycardiologycardiovascular interventionstranslational medicineheart failurebiomedical engineering

Abstract 4144825: 4-Hydroxy-2-Nonenal Alters Alternative Polyadenylation to Regulate Mrna Isoform Diversity in the Transition From Human Cardiac Fibroblasts to Myofibroblasts. Circulation (2024)

Kartiga NatarajanRahul NeupaneHari Krishna YalamanchiliSuresh PalaniyandiEric WagnerAshrith GuhaRajarajan Amirthalingam Thandavarayan

gene functionrna biologyhuman cardiac fibroblastsbiochemistrycellular physiologymrna isoform diversitycell biologymechanobiologyvascular biologytranscription (biology)protein biosynthesisgene expressioncardiovascular researchfibroblast growth factorsstem cell researchrna processingtranscription factorscellular differentiationnucleic acidsmolecular biology

Current Understanding of Cardiovascular Autonomic Dysfunction in Multiple Sclerosis. Heliyon (2024)

Insha ZahoorGuodong PanMirela CerghetTamer ElbayoumiYang Mao-DraayerShailendra GiriSuresh Selvaraj Palaniyandi

neurologycurrent understandinghealth sciencesmultiple sclerosisneurosciencecardiovascular autonomic dysfunction

Maresin-1 Promotes Neuroprotection and Prevents Disease Progression in Experimental Models of Multiple Sclerosis Through Metabolic Reprogramming and Shaping Innate and Adaptive Disease-Associated Cell Types. bioRxiv (Cold Spring Harbor Laboratory) (2023)

Insha ZahoorMohammad NematullahMohammad Ejaz AhmedMena FatmaSajad MirKamesh AyasollaMirela CerghetSuresh PalaniyandiVeronica CeciGiulia CarreraFabio ButtariDiego CentonzeYang Mao-DraayerRamandeep RattanValerio ChiurchiùShailendra Giri

experimental modelsneurologymultiple sclerosismetabolic reprogrammingneuroscienceprevents disease progressionneuroprotection

3′Utr Shortening of Profibrotic Genes and Reversibility of Fibrosis in Patients With End‐Stage Right Ventricular Failure. Clinical and Translational Medicine (2022)

Rahul NeupaneKatarzyna A. CieslikKeith YoukerSuresh Selvaraj PalaniyandiAshrith GuhaRajarajan A. Thandavarayan

gene functionmedicinetranslational researchclinical transplantationliver physiologysurvival analysisclinical researchfibrosisgeneticspharmacologyprofibrotic genescardiovascular researchcardiologymammalian biologymedical geneticsclinical medicinetranslational medicinemedicinal researchclinical therapeutics

Abstract 15145: Aldehyde Dehydrogenase 2 Agonist Reverses Fibroblast Activation in the End Stage Human Right Ventricle Failure via 3’Utr Regulation of Profibrotic Genes. Circulation (2022)

Rahul NeupaneKatarzyna A CieslikKeith YoukerSuresh S PalaniyandiGuha AshrithRajarajan A Thandavarayan

gene functionliver physiologycellular physiologyhuman geneticscardiovascular physiologyapplied physiologytumor biologypharmacologyprofibrotic genesgene expressioninflammationcardiovascular researchcardiologypathway analysisagonist reverseshealth sciencesenzyme inhibitioncardiovascular pharmacologymedical geneticsphysiology

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Suresh Palaniyandi

Assistant Professor

Wayne State University

Affiliations

Graduate School

School of Medicine

Physiology

Suresh Palaniyandi is currently serving as a Graduate School Assistant Professor at Wayne State University.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Suresh Palaniyandi

Wayne State University

Assistant Professor

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Aldh2 and NfκB Activation Restores Vegfr2 Expression and Angiogenesis Impaired by 4‐Hne in Coronary Endothelial Cells. Cell Biochemistry and Function (2026)

inflammationcardiovascular researchfree radical biologyneovascularizationcell signalingblood vesselsvascular biologycell biologyangiogenesisendothelial dysfunctionphysiology

Loss of Hepatic Aldh2 Contributes to Metabolic Stress Mediated Heart Failure. Physiology (2025)

health sciencescardiovascular researchliver physiologymetabolic syndromestress (biology)circulatory physiologydisease pathophysiologyheart failuremetabolismphysiology

neurological progressmultiple sclerosisinflammationmolecular neurodegenerationneuroprotectioncell biologyneuroinflammation

Targeted Overexpression of Mitochondrial Aldh2 in Coronary Endothelial Cells Mitigates Hfpef in a Diabetic Mouse Model. Biomolecules (2025)

mitochondrial functioncardiovascular researchcell metabolismdiabetes researchcell signalingvascular biologycell biologyendothelial dysfunctionphysiology

Therapeutic Strategies for Reversing Cardiac Fibrosis in Hearts Supported by Ventricular Assist Devices. Drug Discovery Today (2025)

pharmacologymedicinedrug discoverydevice therapycardiologycardiovascular interventionstranslational medicineheart failurebiomedical engineering

Abstract 4144825: 4-Hydroxy-2-Nonenal Alters Alternative Polyadenylation to Regulate Mrna Isoform Diversity in the Transition From Human Cardiac Fibroblasts to Myofibroblasts. Circulation (2024)

Kartiga NatarajanRahul NeupaneHari Krishna YalamanchiliSuresh PalaniyandiEric WagnerAshrith GuhaRajarajan Amirthalingam Thandavarayan

Current Understanding of Cardiovascular Autonomic Dysfunction in Multiple Sclerosis. Heliyon (2024)

Insha ZahoorGuodong PanMirela CerghetTamer ElbayoumiYang Mao-DraayerShailendra GiriSuresh Selvaraj Palaniyandi

neurologycurrent understandinghealth sciencesmultiple sclerosisneurosciencecardiovascular autonomic dysfunction

experimental modelsneurologymultiple sclerosismetabolic reprogrammingneuroscienceprevents disease progressionneuroprotection

3′Utr Shortening of Profibrotic Genes and Reversibility of Fibrosis in Patients With End‐Stage Right Ventricular Failure. Clinical and Translational Medicine (2022)

Rahul NeupaneKatarzyna A. CieslikKeith YoukerSuresh Selvaraj PalaniyandiAshrith GuhaRajarajan A. Thandavarayan

Abstract 15145: Aldehyde Dehydrogenase 2 Agonist Reverses Fibroblast Activation in the End Stage Human Right Ventricle Failure via 3’Utr Regulation of Profibrotic Genes. Circulation (2022)

Rahul NeupaneKatarzyna A CieslikKeith YoukerSuresh S PalaniyandiGuha AshrithRajarajan A Thandavarayan

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Similar Faculty Members

Utpal Sen

Institution

University of Louisville

nephrology

renovascular pathophysiology

James West

Institution

Vanderbilt University

idiopathic pulmonary hypertension

pulmonary vascular disease

Ravichandran Ramasamy

Institution

New York University

pharmacology

cardiology

Suresh C. Tyagi

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health science

cardiovascular translational research

Ira J. Goldberg

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metabolism

clinical lipidology

Anindita Das

Institution

Virginia Commonwealth University

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Andrzej S. Tarnawski

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stem cell

biomedical informatics

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fundamental characterization

cardiac ca