Faculty

Institution

Unit

Venue

Lars Dreier

Assistant Professor

University of California, Los Angeles

Affiliations

David Geffen School of Medicine

Biological Chemistry

Lars Dreier is currently serving as a Biological Chemistry Assistant Professor at University of California, Los Angeles.

Honors

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Experiences

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Education

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Lars Dreier

University of California, Los Angeles

Assistant Professor

You can click a keyword to filter the displayed items

neuroscience genetics molecular neuroscience neurology molecular neurobiology neurotransmission molecular biology electrophysiology membrane biology signal transduction neurodegenerative diseases genomics neurogenetics motor control autophagy medicinal research protein science avian biology cellular neurobiology genetic basis mutagenesis amyotrophic lateral sclerosis mitochondrial autophagy voltage-dependent anion channels caenorhabditis elegans locomotor defect synaptic dysfunction synaptic transmission gabaergic motor neurons cell bodies gabaergic synapses cell autophagy cell and molecular biology cell biology cell metabolism

Notable Scholarly Works

Notable Federal Grants

Collaborators

Snb-1-Gfp is Reduced at Gabaergic Synapses and Accumulates in Cell Bodies in Mec-15 Mutants.. (2013)

Sun YuHu ZhitaoGoeb YannickDreier Lars

cell bodiesgeneticsgabaergic synapsesmolecular neuroscienceneurosciencecentral nervous systemneurotransmission

The F-Box Protein Mec-15 (Fbxw9) Promotes Synaptic Transmission in Gabaergic Motor Neurons in C. Elegans. PLoS ONE (2013)

Yu SunZhitao HuYannick GoebLars Dreier

avian biologygeneticssynaptic transmissionneurosciencegabaergic motor neuronsneurologyneurophysiology

Voltage-Dependent Anion Channels (Vdacs) Recruit Parkin to Defective Mitochondria to Promote Mitochondrial Autophagy. Journal of Biological Chemistry (2012)

Yu SunAjay A. VashishtJason TchieuJames A. WohlschlegelLars Dreier

voltage-dependent anion channelscellular neurobiologymolecular neuroscienceneurodegenerative diseaseselectrophysiologyneuroscienceautophagymolecular neurobiologymembrane biologysignal transductionmolecular biologymitochondrial autophagyneurology

An Als-Linked Mutant Sod1 Produces a Locomotor Defect Associated With Aggregation and Synaptic Dysfunction When Expressed in Neurons of Caenorhabditis Elegans. PLoS Genetics (2009)

Jiou WangGeorge W. FarrDavid H. HallFei LiKrystyna FurtakLars DreierArthur L. Horwich

geneticssynaptic dysfunctionmolecular neuroscienceprotein sciencegenetic basisneurosciencegenomicsneurogeneticsmolecular neurobiologycaenorhabditis elegansphysiologymutagenesisneurotransmissionamyotrophic lateral sclerosislocomotor defectmotor controlgenetic engineeringmedicinal research

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Lars Dreier

Assistant Professor

University of California, Los Angeles

Affiliations

David Geffen School of Medicine

Biological Chemistry

Lars Dreier is currently serving as a Biological Chemistry Assistant Professor at University of California, Los Angeles.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Lars Dreier

University of California, Los Angeles

Assistant Professor

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Snb-1-Gfp is Reduced at Gabaergic Synapses and Accumulates in Cell Bodies in Mec-15 Mutants.. (2013)

Sun YuHu ZhitaoGoeb YannickDreier Lars

cell bodiesgeneticsgabaergic synapsesmolecular neuroscienceneurosciencecentral nervous systemneurotransmission

The F-Box Protein Mec-15 (Fbxw9) Promotes Synaptic Transmission in Gabaergic Motor Neurons in C. Elegans. PLoS ONE (2013)

Yu SunZhitao HuYannick GoebLars Dreier

avian biologygeneticssynaptic transmissionneurosciencegabaergic motor neuronsneurologyneurophysiology

Voltage-Dependent Anion Channels (Vdacs) Recruit Parkin to Defective Mitochondria to Promote Mitochondrial Autophagy. Journal of Biological Chemistry (2012)

Yu SunAjay A. VashishtJason TchieuJames A. WohlschlegelLars Dreier

An Als-Linked Mutant Sod1 Produces a Locomotor Defect Associated With Aggregation and Synaptic Dysfunction When Expressed in Neurons of Caenorhabditis Elegans. PLoS Genetics (2009)

Jiou WangGeorge W. FarrDavid H. HallFei LiKrystyna FurtakLars DreierArthur L. Horwich

1
10 / page

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Similar Faculty Members

Albert Russell La Spada

Institution

University of California, Irvine

neurogenetics

neuroscience

Maureen McCall

Institution

University of Louisville

ophthalmology

ophthalmology retina

Lawrence Goldstein

Institution

University of California, San Diego

developmental system

developmental neurobiology

Colin G. Nichols

Institution

Washington University in St. Louis

membrane transport

electrophysiology

Steven J. Burden, PhD

Institution

New York University

brain aging

molecular neurobiology

Aaron DiAntonio

Institution

Washington University in St. Louis

degenerative pathology

neurogenetics

Ta Yuan Chang

Institution

Dartmouth College

autophagy

obesity

Akira Sawa

Institution

Johns Hopkins University

neuroscience

psychiatry

Sushil Mahata

Institution

University of California, San Diego

metabolic disorder

hormone secretion

Gabriel G. Haddad

Institution

University of California, San Diego

respiratory medicine

pediatric pulmonology