Faculty

Institution

Unit

Venue

Fei Chen

Professor

Stony Brook University

Affiliations

Renaissance School of Medicine

Pathology

Fei Chen is currently serving as a Pathology Professor at Stony Brook University.

Honors

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Experiences

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Education

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Fei Chen

Stony Brook University

Professor

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oncology cancer research cancer biology tumor biology cell biology molecular biology gene expression cancer genetics pharmacology medicinal research genetics carcinogenesis oncogenes molecular cancer toxicology redox biology cell signaling epigenetics cellular therapy enzyme inhibition vascular biology molecular cell biology tumor targeting redox signaling cancer genomics cancer discovery cell research cancer cells genome biology oncogenic agent tumor suppressors epigenetic changes cancer stem cells stem cell research mammalian biology brain tumor research liver physiology cancer treatment toxicological mechanism protein phosphorylation cancer risk anti-cancer agents cell regulation cell migration gene regulation medical genetics cancer growth cell physiology liver cancer clinical toxicology cell cycle arsenic-induced carcinogenesis glioblastoma cells environmental arsenic-induced carcinogenesis cell therapy key site inhibitory activity apoptosis uneven development viral oncology in vivo systems stem-cell therapy metabolic remodeling genomic medicine cancer therapy compensatory growth jnk-induced apoptosis arsenic-induced akt activation arsenic exposure lung cancer cells gefitinib resistance epigenetic reprogramming silica-induced pulmonary fibrosis mineral dust selenite uptake increased expression cancer letter histone methylation epigenomics pathology

Notable Scholarly Works

Notable Federal Grants

Collaborators

Nrf2 Orchestrates Epigenetic Regulations and Serves as the Master Regulator of Klf4 Expression and Activity During Arsenic-Induced Transformation. bioRxiv (Cold Spring Harbor Laboratory) (2024)

Ziwei WangZhuoyue BiJessica BamrahYiran QiuWenxuan ZhangHaoyan JiJohn D. HaleyChitra ThakurFei Chen

master regulatordna repairbiochemistryoxidative stressprotein sciencenitrosative stressepigeneticsmolecular cell biologygene expressionfree radical biologyepigenetic regulationstranscription factorsenvironmental epigeneticsenzyme inhibitionmolecular biologysystems biologyepigenomicsepigenetic changesarsenic-induced transformationtrace elements

Tumor Suppressive Activity of Ahr in Environmental Arsenic-Induced Carcinogenesis. Toxicology and Applied Pharmacology (2023)

Ziwei WangYao FuAkimasa SenoZhuoyue BiAashna S. PawarHaoyan JiBandar Saeed AlmutairyYiran QiuWenxuan ZhangChitra ThakurFei Chen

neurologypharmacologytoxicologyapplied pharmacologymedicinetumor suppressorsenvironmental toxicologycancer biologycancer researchanalytical toxicologyoncologyenvironmental arsenic-induced carcinogenesismetal toxicitypharmacotherapyanti-cancer agentstumor suppressive activitycarcinogenesisclinical toxicology

Arsenic Activates Stat3 Signaling During the Transformation of the Human Bronchial Epithelial Cells. Toxicology and Applied Pharmacology (2022)

Bandar AlmutairyYao FuZhuoyue BiWenxuan ZhangPriya WadgaonkarYiran QiuChitra ThakurFei Chen

pharmacologynanomedicinetoxicologyapplied pharmacologygene expressionhealth sciencesmedicineexperimental toxicologyrespiratory toxicologyredox signalingarsenic activatesenzyme inhibitionredox biologypharmacotherapyclinical toxicologyhematologycatalyst activation

Arsenic Activates the Er Stress-Associated Unfolded Protein Response via the Activating Transcription Factor 6 in Human Bronchial Epithelial Cells. Biomedicines (2022)

Priya WadgaonkarZhuoyue BiJunmei WanYao FuQian ZhangBandar AlmutairyWenxuan ZhangYiran QiuChitra ThakurMaik HüttemannFei Chen

pharmacologybiomedical researchmembrane biologymedicineliver physiologytranscription factorsprotein foldingheme homeostasisprotein misfoldingredox biologyclinical medicineredox signalingclinical researchtranslational medicinemedicinal researchclinical biochemistrycell biologyvascular biology

Card9 Mediates Pancreatic Islet Beta-Cell Dysfunction Under the Duress of Hyperglycemic Stress. Cellular Physiology and Biochemistry (2022)

Suhadinie GamageMirabela HaliFei ChenAnjaneyulu Kowluru

membrane biologyendocrinologycellular biochemistryhyperglycemic stressmolecular physiologybiochemistrycellular physiologycell physiologycell biologycardiovascular diseasehuman physiologyelectrophysiologytissue physiologyphysiology

Deletion of Mdig Enhances H3K36Me3 and Metastatic Potential of the Triple Negative Breast Cancer Cells. iScience (2022)

Chitra ThakurYiran QiuQian ZhangNicholas J. CarruthersMiaomiao YuZhuoyue BiYao FuPriya WadgaonkarBandar AlmutairyAkimasa SenoPaul M. StemmerFei Chen

pathologytumor targetingcancer cellscell biologycancer biologycancer researchmetastasistumor biologymetastatic potentialmolecular cell biologycancer treatmentbreast canceroncologycellular therapycancer geneticsoncogenesmolecular biologymedicinal researchcancer growth

Depletion of Mdig Changes Proteomic Profiling in Triple Negative Breast Cancer Cells. Biomedicines (2022)

Chitra ThakurNicholas J. CarruthersQian ZhangLiping XuYao FuZhuoyue BiYiran QiuWenxuan ZhangPriya WadgaonkarBandar AlmutairyChunna GuoPaul M. StemmerFei Chen

medicineclinical researchcell biologyprotein sciencecancer biologybiomedical researchcancer researchproteomicscancer genomicstumor biologybreast cancerbreast oncologyclinical proteomicsdrug analysismdig changesoncogenesclinical medicinemolecular biologymedicinal research

Connections Between Endoplasmic Reticulum Stress-Associated Unfolded Protein Response, Mitochondria, and Autophagy in Arsenic-Induced Carcinogenesis. Seminars in Cancer Biology (2021)

Priya WadgaonkarFei Chen

arsenic-induced carcinogenesiscancer biologyautophagymolecular biologycarcinogenesis

Cooperation Between Nrf2-Mediated Transcription and Mdig-Dependent Epigenetic Modifications in Arsenic-Induced Carcinogenesis and Cancer Stem Cells. Seminars in Cancer Biology (2021)

Zhuoyue BiQian ZhangYao FuAkimasa SenoPriya WadgaonkarYiran QiuBandar AlmutairyLiping XuWenxuan ZhangChitra ThakurFei Chen

geneticscancer biologymolecular cancercancer researchcancer genomicstumor biologycarcinogenesisepigeneticsarsenic-induced carcinogenesiscancer stem cellsmolecular cell biologyoncologyredox biologycancer geneticscancer carecancer riskcancer managementmdig-dependent epigenetic modificationsoncogenesradiation oncologymolecular biology

Design and Synthesis of Isothiocyanate‐Containing Hybrid Androgen Receptor (Ar) Antagonist to Downregulate Ar and Induce Ferroptosis in Gsh–Deficient Prostate Cancer Cells. Chemical Biology & Drug Design (2021)

Zhihui QinSiyu OuLiping XuKathleen SorensenYingxue ZhangDan‐Ping HuZhe YangWen‐Yang HuFei ChenGail S. Prins

rational drug designpharmacologyurologyreceptorstumor targetingdrug discoverypeptide sciencereceptor (biochemistry)pharmaceutical researchpharmaceutical sciencesenzyme inhibitioncellular therapycell signalingpeptidesmedicinal researchcell biologychemical biologyprostate cancer

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Fei Chen

Professor

Stony Brook University

Affiliations

Renaissance School of Medicine

Pathology

Fei Chen is currently serving as a Pathology Professor at Stony Brook University.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Fei Chen

Stony Brook University

Professor

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Nrf2 Orchestrates Epigenetic Regulations and Serves as the Master Regulator of Klf4 Expression and Activity During Arsenic-Induced Transformation. bioRxiv (Cold Spring Harbor Laboratory) (2024)

Ziwei WangZhuoyue BiJessica BamrahYiran QiuWenxuan ZhangHaoyan JiJohn D. HaleyChitra ThakurFei Chen

Tumor Suppressive Activity of Ahr in Environmental Arsenic-Induced Carcinogenesis. Toxicology and Applied Pharmacology (2023)

Ziwei WangYao FuAkimasa SenoZhuoyue BiAashna S. PawarHaoyan JiBandar Saeed AlmutairyYiran QiuWenxuan ZhangChitra ThakurFei Chen

Arsenic Activates Stat3 Signaling During the Transformation of the Human Bronchial Epithelial Cells. Toxicology and Applied Pharmacology (2022)

Bandar AlmutairyYao FuZhuoyue BiWenxuan ZhangPriya WadgaonkarYiran QiuChitra ThakurFei Chen

Arsenic Activates the Er Stress-Associated Unfolded Protein Response via the Activating Transcription Factor 6 in Human Bronchial Epithelial Cells. Biomedicines (2022)

Priya WadgaonkarZhuoyue BiJunmei WanYao FuQian ZhangBandar AlmutairyWenxuan ZhangYiran QiuChitra ThakurMaik HüttemannFei Chen

Card9 Mediates Pancreatic Islet Beta-Cell Dysfunction Under the Duress of Hyperglycemic Stress. Cellular Physiology and Biochemistry (2022)

Suhadinie GamageMirabela HaliFei ChenAnjaneyulu Kowluru

Deletion of Mdig Enhances H3K36Me3 and Metastatic Potential of the Triple Negative Breast Cancer Cells. iScience (2022)

Chitra ThakurYiran QiuQian ZhangNicholas J. CarruthersMiaomiao YuZhuoyue BiYao FuPriya WadgaonkarBandar AlmutairyAkimasa SenoPaul M. StemmerFei Chen

Depletion of Mdig Changes Proteomic Profiling in Triple Negative Breast Cancer Cells. Biomedicines (2022)

Chitra ThakurNicholas J. CarruthersQian ZhangLiping XuYao FuZhuoyue BiYiran QiuWenxuan ZhangPriya WadgaonkarBandar AlmutairyChunna GuoPaul M. StemmerFei Chen

Connections Between Endoplasmic Reticulum Stress-Associated Unfolded Protein Response, Mitochondria, and Autophagy in Arsenic-Induced Carcinogenesis. Seminars in Cancer Biology (2021)

Priya WadgaonkarFei Chen

arsenic-induced carcinogenesiscancer biologyautophagymolecular biologycarcinogenesis

Cooperation Between Nrf2-Mediated Transcription and Mdig-Dependent Epigenetic Modifications in Arsenic-Induced Carcinogenesis and Cancer Stem Cells. Seminars in Cancer Biology (2021)

Zhuoyue BiQian ZhangYao FuAkimasa SenoPriya WadgaonkarYiran QiuBandar AlmutairyLiping XuWenxuan ZhangChitra ThakurFei Chen

Zhihui QinSiyu OuLiping XuKathleen SorensenYingxue ZhangDan‐Ping HuZhe YangWen‐Yang HuFei ChenGail S. Prins

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