Faculty

Institution

Unit

Venue

Zachary Weil

Associate Professor

West Virginia University

Affiliations

School of Medicine

Neuroscience

Zachary Weil is currently serving as a Neuroscience Associate Professor at West Virginia University.

Honors

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Experiences

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Education

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Zachary Weil

West Virginia University

Associate Professor

You can click a keyword to filter the displayed items

neuroscience neurology mammalian biology physiology behavioral neuroscience endocrinology immunology avian biology cognitive neuroscience medicinal research animal physiology neurophysiology animal behavior brain science chronobiology traumatic brain injury mammalian physiology neural science sleep inflammation neuropsychology dim light neuroanatomy affective neuroscience biological psychiatry clinical neuroscience siberian hamsters animal science animal cognition brain-immune interaction livestock science psychoneuroendocrinology biological psychology behavioral neuroendocrinology neuropsychopharmacology comparative endocrinology endocrine research brain function animal ecology molecular psychiatry rodent ecology comparative physiology neuroinflammation animal reproduction breeding behavior photobiology cytokines melatonin brain health immune function male siberian hamsters neurorehabilitation behavioral research human sciences human physiology cellular neurobiology host response alcohol misuse behavior analysis depressive-like responses neuronal morphology traumatic brain injuries integrative biology alcohol abuse movement ecology biochronology cognitive psychology neuroprotection male mice affective responses inflammatory responses sleep deprivation male white-footed mice diurnal rhythm hippocampal neuronal morphology cell-mediated immunity functional recovery cognitive rehabilitation ischemic syndrome cerebral blood flow brain injury trauma stroke head trauma rehabilitation

Notable Scholarly Works

Notable Federal Grants

Collaborators

Amino Acid Compound 2 (Aac2) Treatment Counteracts Insulin-Induced Synaptic Gene Expression and Seizure-Related Mortality in a Mouse Model of Alzheimer’s Disease. International Journal of Molecular Sciences (2024)

Zhijie DengAejin LeeTao LinSagarika TanejaDevan KowdleyJacob H. LeungMarykate HillTianyi TaoJulie FitzgeraldLianbo YuJoshua J. BlakesleeKristy TownsendZachary M. WeilJon R. ParquetteOuliana Ziouzenkova

mouse modelpeptidesmolecular scienceneurochemistrymolecular neurobiologyneuropharmacologyseizure-related mortalitymolecular biologymolecular neurodegenerationneurophysiologymedicinal researchalzheimer's diseaseneuroscienceneurodegenerative diseasesbiochemistryepilepsy

Author Response for"Cerebral Hypoperfusion Exacerbates Traumatic Brain Injury in Male but Not Female Mice". (2024)

None Bailey J. WhiteheadNone Deborah CorbinNone Megan L. AlexanderNone Jacob BumgarnerNone Ning ZhangNone Kate KarelinaNone Zachary M. Weil

neurologymedicineauthor responsetraumatic brain injuryveterinary medicinemammalian biologycerebral hypoperfusionneurosciencefemale mice

Cerebral Hypoperfusion Exacerbates Traumatic Brain Injury in Male but Not Female Mice. European Journal of Neuroscience (2024)

Bailey J. WhiteheadDeborah CorbinMegan L. AlexanderJacob BumgarnerNing ZhangKate KarelinaZachary M. Weil

cognitive neuroscienceneurologytraumatic brain injuryclinical neurosciencemammalian biologycerebral hypoperfusionneural scienceneuroscience

Cerebral Hypoperfusion Exacerbates Vascular Dysfunction After Traumatic Brain Injury. Experimental Neurology (2024)

Bailey WhiteheadDeborah CorbinEthan MeadowsNing ZhangJohn M. HollanderKate KarelinaZachary M. Weil

neurologyvascular dysfunctionemergency medicinetraumatic brain injurynephrologyneurosciencecerebral hypoperfusion

Neuronal Bag3 Attenuates Tau Hyperphosphorylation, Synaptic Dysfunction, and Cognitive Deficits Induced by Traumatic Brain Injury via the Regulation of Autophagy-Lysosome Pathway. Acta Neuropathologica (2024)

Nicholas SweeneyTae Yeon KimCody T. MorrisonLiangping LiDiana AcostaJiawen LiangNithin V. DatlaJulie A. FitzgeraldHaoran HuangXianglan LiuGregory Huang TanMin WuKate KarelinaChelsea E. BrayZachary M. WeilDouglas W. ScharreGeidy E. SerranoTakashi SaitoTakaomi C. SaidoThomas G. BeachOlga N. Kokiko-CochranJonathan P. GodboutGail V. W. JohnsonHongjun Fu

cognitive neuroscienceneurologybrain injurytau hyperphosphorylationtraumatic brain injuryautophagy-lysosome pathwayneuropathologysynaptic dysfunctionexperimental neuropathologycognitive deficitsbrain functionautophagyneuroscienceneurodegenerative diseases

Exercise Intensity and Sex Alter Neurometabolic, Transcriptional, and Functional Recovery Following Traumatic Brain Injury. Experimental Neurology (2023)

Brishti A. WhiteJulia T. IveyRuth Velazquez-CruzRobin OliverioBailey WhiteheadMark PintiJohn HollanderLi MaGangquin HuZachary M. WeilKate Karelina

physiologyneurologycognitive neurosciencefunctional recoveryexercise intensitytraumatic brain injuryexercise physiologybrain functionneuroscienceneurophysiology

Mild Traumatic Brain Injury Induces Pericyte Detachment Independent of Stroke Vulnerability. Neuroscience Letters (2023)

Bailey WhiteheadDeborah CorbinAli AlbowaideyNing ZhangKate KarelinaZachary M. Weil

strokeneurologystroke vulnerabilityneuroscience

Pericyte Dysfunction is a Key Mediator of the Risk of Cerebral Ischemia. Journal of Neuroscience Research (2023)

Bailey WhiteheadKate KarelinaZachary M. Weil

brain scienceneurologykey mediatorvascular biologyclinical neurosciencebrain healthcerebral ischemiastrokepericyte dysfunctionneurosciencehealth sciences

Arnold Adolph Berthold. Springer eBooks (2022)

Zachary M. WeilRandy J. Nelson

medicineliterature

Bag3 Attenuates Tau Hyperphosphorylation and Gliosis Induced by Traumatic Brain Injury. Alzheimer s & Dementia (2022)

Cody MorrisonLiangping LiJiawen LiangShuo ChenDiana M AcostaJulie A FitzgeraldKate KarelinaChelsea E BrayZachary M WeilJonathan GodboutOlga N Kokiko‐cochranTakashi SaitoTakaomi C. SaidoHongjun Fu

cognitive neuroscienceneurologytau hyperphosphorylationtraumatic brain injurydementiaalzheimer's diseaseneuroscience

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Zachary Weil

Associate Professor

West Virginia University

Affiliations

School of Medicine

Neuroscience

Zachary Weil is currently serving as a Neuroscience Associate Professor at West Virginia University.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Zachary Weil

West Virginia University

Associate Professor

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Author Response for"Cerebral Hypoperfusion Exacerbates Traumatic Brain Injury in Male but Not Female Mice". (2024)

None Bailey J. WhiteheadNone Deborah CorbinNone Megan L. AlexanderNone Jacob BumgarnerNone Ning ZhangNone Kate KarelinaNone Zachary M. Weil

neurologymedicineauthor responsetraumatic brain injuryveterinary medicinemammalian biologycerebral hypoperfusionneurosciencefemale mice

Cerebral Hypoperfusion Exacerbates Traumatic Brain Injury in Male but Not Female Mice. European Journal of Neuroscience (2024)

Bailey J. WhiteheadDeborah CorbinMegan L. AlexanderJacob BumgarnerNing ZhangKate KarelinaZachary M. Weil

cognitive neuroscienceneurologytraumatic brain injuryclinical neurosciencemammalian biologycerebral hypoperfusionneural scienceneuroscience

Cerebral Hypoperfusion Exacerbates Vascular Dysfunction After Traumatic Brain Injury. Experimental Neurology (2024)

Bailey WhiteheadDeborah CorbinEthan MeadowsNing ZhangJohn M. HollanderKate KarelinaZachary M. Weil

neurologyvascular dysfunctionemergency medicinetraumatic brain injurynephrologyneurosciencecerebral hypoperfusion

Exercise Intensity and Sex Alter Neurometabolic, Transcriptional, and Functional Recovery Following Traumatic Brain Injury. Experimental Neurology (2023)

Brishti A. WhiteJulia T. IveyRuth Velazquez-CruzRobin OliverioBailey WhiteheadMark PintiJohn HollanderLi MaGangquin HuZachary M. WeilKate Karelina

physiologyneurologycognitive neurosciencefunctional recoveryexercise intensitytraumatic brain injuryexercise physiologybrain functionneuroscienceneurophysiology

Mild Traumatic Brain Injury Induces Pericyte Detachment Independent of Stroke Vulnerability. Neuroscience Letters (2023)

Bailey WhiteheadDeborah CorbinAli AlbowaideyNing ZhangKate KarelinaZachary M. Weil

strokeneurologystroke vulnerabilityneuroscience

Pericyte Dysfunction is a Key Mediator of the Risk of Cerebral Ischemia. Journal of Neuroscience Research (2023)

Bailey WhiteheadKate KarelinaZachary M. Weil

brain scienceneurologykey mediatorvascular biologyclinical neurosciencebrain healthcerebral ischemiastrokepericyte dysfunctionneurosciencehealth sciences

Arnold Adolph Berthold. Springer eBooks (2022)

Zachary M. WeilRandy J. Nelson

medicineliterature

Bag3 Attenuates Tau Hyperphosphorylation and Gliosis Induced by Traumatic Brain Injury. Alzheimer s & Dementia (2022)

Cody MorrisonLiangping LiJiawen LiangShuo ChenDiana M AcostaJulie A FitzgeraldKate KarelinaChelsea E BrayZachary M WeilJonathan GodboutOlga N Kokiko‐cochranTakashi SaitoTakaomi C. SaidoHongjun Fu

cognitive neuroscienceneurologytau hyperphosphorylationtraumatic brain injurydementiaalzheimer's diseaseneuroscience

1
2
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4
5
17
10 / page

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stress responsive systems

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Emory University

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