Faculty

Institution

Unit

Venue

Thomas Ma

Professor

Pennsylvania State University

Affiliations

Penn State College of Medicine

Department of Cellular & Molecular Physiology

Department of Medicine

Department of Microbiology and Immunology

Department of Medicine

Division of Gastroenterology and Hepatology

Thomas Ma is currently serving as a Cellular & Molecular Physiology Professor at Pennsylvania State University.

Honors

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Experiences

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Education

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Thomas Ma

Pennsylvania State University

Professor

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gastroenterology membrane biology cell biology physiology inflammation clinical microbiology inflammatory bowel disease digestive diseases neuroscience vascular biology hepatology autophagy mucosal immunology cellular physiology pathway analysis biophysics veterinary medicine regenerative medicine molecular physiology clinical immunology digestive tract tissue (biology) liver physiology matrix biology mammalian biology applied physiology medicinal research redox biology cytokines cell-matrix interactions translational physiology cell physiology tissue physiology cellular biology molecular regulation clinical physiology disease physiology microbial physiology clinical allergy molecular immunology experimental colitis intestinal permeability proton pump inhibitors circulatory physiology protein science sociobiology cell research cell biomechanics cutaneous biology cell regulation cancer biology veterinary microbiology medical microbiology cell interactions catalyst activation veterinary physiology drug allergy patient satisfaction microbial genetics physiological research organ physiology gene regulation gene function cytokine modulation macromolecule flux experimental dss colitis autophagy-related protein epithelial cell junctions paracellular macromolecule flux membrane localization dss-induced murine colitis key factor long-term use intestinal inflammation inflammation-inducing cytokines aryl hydrocarbon receptor molecular gastroenterology intestinal barrier gut-organ axis dysbiosis infectious diseases microbiome immunology pathogenesis gut barrier

Notable Scholarly Works

Notable Federal Grants

Collaborators

Bifidobacterium Bifidum Inhibits the Tnf-α Induced Increase in Intestinal Epithelial Tight Junction Permeability via a Tlr-2/Tlr-6 and Ppar-γ Dependent Inhibition of Nf-κB P50/P65. Physiology (2024)

Raz AbdulqadirRana Al-SadiThomas Ma

immunologyredox biologybacteriapharmacologyclinical microbiologyvascular biologygastroenterologymicrobial physiologyenzyme inhibitiongene expressiontnf-a induced increaseinhibitory activitydigestive diseasesmicrobiologyppar-g dependent inhibitionhost specificitydisease physiologyinflammationmembrane biologyhost factors

Alpha-Tocopherylquinone Differentially Modulates Claudins to Enhance Intestinal Epithelial Tight Junction Barrier via Ahr and Nrf2 Pathways. Cell Reports (2023)

Ashwinkumar Subramenium GanapathyKushal SahaAlexandra WangPriya ArumugamViszwapriya DharmaprakashGregory YochumWalter KoltunMeghali NighotGary PerdewTodd A. ThompsonThomas MaPrashant Nighot

cell physiologyvascular biologycell interactionsphysiologycell signalingcell researchcell biologyhepatologycellular physiologymedicinal researchmembrane biologycellular biologypathway analysispharmacologydigestive tractsignal transductiongene expressionredox biology

Alpha-Tocopherylquinone-Mediated Activation of the Aryl Hydrocarbon Receptor Regulates the Production of Inflammation-Inducing Cytokines and Ameliorates Intestinal Inflammation. Mucosal Immunology (2023)

Kushal SahaAshwinkumar Subramenium GanapathyAlexandra WangPriya ArumugamNathan Michael MorrisLeonard HarrisGregory YochumWalter KoltunGary H. PerdewMeghali NighotThomas MaPrashant Nighot

inflammationalpha-tocopherylquinone-mediated activationinflammation-inducing cytokinesimmunologyaryl hydrocarbon receptormucosal immunologypharmacologyintestinal inflammationcytokines

Bifidobacterium Bifidum Causes an Enhancement of the Intestinal Epithelial Tight Junction Barrier is Mediated by Tlr-2 Dependent Increases of Toll-Interacting Protein (Tollip) Expression in a Myd88 Independent Manner. Physiology (2023)

Raz AbdulqadirRana Al-SadiThomas Ma

organ physiologyimmunologymicrobiomeclinical microbiologyphysiological researchphysiologytoll-interacting proteinmicrobial geneticsapplied physiologygene expressionreceptorscellular physiologymicrobiologypathway analysisbifidobacterium bifidumapplied microbiologyelectrophysiologymolecular physiologymembrane biologyclinical physiology

Bifidobacterium Bifidum Prevents the Il-1B Induced Increase in Intestinal Permeability by a Novel Mechanism: Tlr-2 Dependent Activation of Ppar-Gamma and Inhibition of Nf-Kb Signaling Pathway. Physiology (2023)

Raz AbdulqadirRana Al-SadiThomas Ma

clinical microbiologyinflammationvascular biologyphysiologygastroenterologyimmunologymicrobial physiologymembrane biologyimmune responsesclinical physiologypharmacologybifidobacterium bifidumcatalyst activationveterinary medicineintestinal permeabilitynovel mechanismveterinary physiologydisease physiology

Lactobacillus Acidophilus Induces Enhancement of the Intestinal Epithelial Tight Junction Barrier Function by Toll-Like Receptor-2 Independent of Myd88. Journal of Allergy and Clinical Immunology (2023)

Lauren KaminskyRana Al-SadiJessica EngersThomas Ma

clinical microbiologyinflammationmolecular immunologyimmunologymedicinal researchclinical allergyallergyimmune responsespathway analysispharmacologymucosal immunologybiomarkersreceptorsclinical immunology

Autophagy Increases Occludin Levels to Enhance Intestinal Paracellular Tight Junction Barrier. bioRxiv (Cold Spring Harbor Laboratory) (2022)

Kushal SahaAshwinkumar Subramenium GanapathyAlexandria WangNathan Michael MorrisEric SuchanecGregory YochumWalter KoltunWei DingMeghali NighotThomas MaPrashant Nighot

membrane biologyregenerative medicinecell biologyautophagy

Autophagy Reduces the Degradation and Promotes Membrane Localization of Occludin to Enhance the Intestinal Epithelial Tight Junction Barrier Against Paracellular Macromolecule Flux. Journal of Crohn s and Colitis (2022)

Kushal SahaAshwinkumar Subramenium GanapathyAlexandra WangNathan Michael MorrisEric SuchanecWei DingGregory YochumWalter KoltunMeghali NighotThomas MaPrashant Nighot

vascular biologymembrane localizationinflammatory bowel diseaseautophagyparacellular macromolecule fluxhealth sciences

Lactobacillus Acidophilus Causes Enhancement of the Intestinal Tight Junction Barrier by a Toll-Like Receptor-2-Dependent Increase in Occludin. Journal of Allergy and Clinical Immunology (2022)

Lauren KaminskyRana Al-SadiThomas Ma

clinical microbiologyinflammationmolecular immunologyimmunologyclinical allergyallergyimmune responsespathway analysispharmacologyreceptorsdrug allergyclinical immunology

Long-Term Use of Proton Pump Inhibitors Disrupts Intestinal Tight Junction Barrier and Exaggerates Experimental Colitis. Journal of Crohn s and Colitis (2022)

Meghali NighotPei-Luan LiaoNathan MorrisDennis McCarthyViszwapriya DharmaprakashInam Ullah KhanShannon DalessioKushal SahaAshwinkumar Subramaniam GanapathyAlexandra WangWei DingGregory YochumWalter KoltunPrashant NighotThomas Ma

neurologyexperimental colitisgastroenterologyrespiratory medicineinflammatory bowel diseaseproton pump inhibitorspharmacologylong-term useveterinary medicinehealth sciences

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Thomas Ma

Professor

Pennsylvania State University

Affiliations

Penn State College of Medicine

Department of Cellular & Molecular Physiology

Department of Medicine

Department of Microbiology and Immunology

Department of Medicine

Division of Gastroenterology and Hepatology

Thomas Ma is currently serving as a Cellular & Molecular Physiology Professor at Pennsylvania State University.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Thomas Ma

Pennsylvania State University

Professor

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Raz AbdulqadirRana Al-SadiThomas Ma

Alpha-Tocopherylquinone Differentially Modulates Claudins to Enhance Intestinal Epithelial Tight Junction Barrier via Ahr and Nrf2 Pathways. Cell Reports (2023)

Ashwinkumar Subramenium GanapathyKushal SahaAlexandra WangPriya ArumugamViszwapriya DharmaprakashGregory YochumWalter KoltunMeghali NighotGary PerdewTodd A. ThompsonThomas MaPrashant Nighot

Kushal SahaAshwinkumar Subramenium GanapathyAlexandra WangPriya ArumugamNathan Michael MorrisLeonard HarrisGregory YochumWalter KoltunGary H. PerdewMeghali NighotThomas MaPrashant Nighot

inflammationalpha-tocopherylquinone-mediated activationinflammation-inducing cytokinesimmunologyaryl hydrocarbon receptormucosal immunologypharmacologyintestinal inflammationcytokines

Raz AbdulqadirRana Al-SadiThomas Ma

Lauren KaminskyRana Al-SadiJessica EngersThomas Ma

Autophagy Increases Occludin Levels to Enhance Intestinal Paracellular Tight Junction Barrier. bioRxiv (Cold Spring Harbor Laboratory) (2022)

Kushal SahaAshwinkumar Subramenium GanapathyAlexandria WangNathan Michael MorrisEric SuchanecGregory YochumWalter KoltunWei DingMeghali NighotThomas MaPrashant Nighot

membrane biologyregenerative medicinecell biologyautophagy

Kushal SahaAshwinkumar Subramenium GanapathyAlexandra WangNathan Michael MorrisEric SuchanecWei DingGregory YochumWalter KoltunMeghali NighotThomas MaPrashant Nighot

vascular biologymembrane localizationinflammatory bowel diseaseautophagyparacellular macromolecule fluxhealth sciences

Lactobacillus Acidophilus Causes Enhancement of the Intestinal Tight Junction Barrier by a Toll-Like Receptor-2-Dependent Increase in Occludin. Journal of Allergy and Clinical Immunology (2022)

Lauren KaminskyRana Al-SadiThomas Ma

clinical microbiologyinflammationmolecular immunologyimmunologyclinical allergyallergyimmune responsespathway analysispharmacologyreceptorsdrug allergyclinical immunology

Long-Term Use of Proton Pump Inhibitors Disrupts Intestinal Tight Junction Barrier and Exaggerates Experimental Colitis. Journal of Crohn s and Colitis (2022)

neurologyexperimental colitisgastroenterologyrespiratory medicineinflammatory bowel diseaseproton pump inhibitorspharmacologylong-term useveterinary medicinehealth sciences

1
2
3
10 / page

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Similar Faculty Members

Venkatakrishna Jala

Institution

University of Louisville

chemokine receptors

dysbiosis

Waddah Alrefai

Institution

University of Illinois Chicago

gastroenterology

physiology

Christian Jobin

Institution

University of Florida

innate immune system

pathology

Terrence Barrett

Institution

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Gary Wu

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metabolism

host metabolism

Peter Ernst

Institution

University of California, San Diego

celiac disease

allergy

Adam Moeser

Institution

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transgenerational effect

biological sex

Lars Eckmann

Institution

University of California, San Diego

innate immune system

gut barrier

Pawel Kiela

Institution

University of Arizona

mucosal immunology

immunology

Andrew D. Patterson

Institution

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health science

dysbiosis