Faculty

Institution

Unit

Venue

Tariq Haqqi

Northeast Ohio Medical University

Affiliations

Department Of Cardiovascular Medicine

Northeast Ohio Medical University

NoneTariq Haqqi is currently serving as a Cardiovascular Medicine faculty at Northeast Ohio Medical University.

Honors

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Experiences

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Education

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Tariq Haqqi

Northeast Ohio Medical University

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immunology osteoarthritis rheumatology inflammation cell biology pharmacology cartilage regenerative medicine medicine musculoskeletal research gene expression arthritis orthopaedic research cell signaling orthopedic surgery bone research anti-inflammatory oxidative stress medicinal research microrna detection cytokines experimental medicine clinical rheumatology musculoskeletal disorders inflammatory diseases human chondrocytes free radical biology chronic inflammation human oa chondrocytes cellular physiology redox signaling herbal medicine bone homeostasis bone disease inflammatory rheumatic disease rna biology redox biology vascular biology protein science tissue physiology apoptosis innate immune system human osteoarthritis chondrocytes primary human chondrocytes pharmacotherapy liver physiology pharmacological research plant immunity skeletal muscle mitochondrial function cell physiology cellular biology radical (chemistry) protein function human tissues clinical immunology natural flavonoid polyphenolics immunopathology disease progression receptor tyrosine kinases heme signaling cell fate determination functional modification medical nutrition osteoarthritis pathogenesis chondroprotective effects parkin clearance dysfunctional mitochondria ros levels mitogen-activated protein kinases nuclear factor- b therapeutic aspects apoptotic response human articular chondrocytes nf- b

Notable Scholarly Works

Notable Federal Grants

Collaborators

Erk1/2-Mediated Activation of Drp1 Regulates Mitochondrial Dynamics and Apoptosis in Chondrocytes. Osteoarthritis and Cartilage (2021)

M Y AnsariK NovakT M Haqqi

regenerative medicineapoptosisrheumatologyphysiologycell deathosteoarthritiscell signalingcell biologyorthopaedic researchneuroscienceorthopedic surgeryprotein functionbone diseasemusculoskeletal researchheme signalingmusculoskeletal disorderscartilagebone research

A Retrotransposon Gag-Like-3 Gene Rtl3 and Sox-9 Co-Regulate the Expression of Col2A1 in Chondrocytes. Connective Tissue Research (2020)

connective tissue diseasecell biologyneurosciencetissue physiologyhuman tissuestranscription (biology)geneticssignal transductiongene expressiontissue (biology)tissue engineeringbone research

Assessing Chondrocyte Status by Immunofluorescence-Mediated Localization of Parkin Relative to Mitochondria. Methods in molecular biology (2020)

osteoarthritisparkin relativecell biologycellular physiologybiochemistrymusculoskeletal tissuesmusculoskeletal researchchondrocyte statusimmunofluorescence-mediated localizationextracellular matrixtissue (biology)cartilage

Chondrocytes. Methods in molecular biology (2020)

Imperatorin Suppresses Il-1β-Induced Inos Expression via Inhibiting Erk-Mapk/Ap1 Signaling in Primary Human Oa Chondrocytes. International Immunopharmacology (2020)

Nashrah AhmadMohammad Y. AnsariShabana BanoTariq M Haqqi

immunologypharmacologydermatology

Lysosomal Dysfunction in Osteoarthritis and Aged Cartilage Triggers Apoptosis in Chondrocytes Through Bax Mediated Release of Cytochrome C. Osteoarthritis and Cartilage (2020)

M.Y. AnsariH.C. BallS.J. WaseK. NovakT.M. Haqqi

inflammationapoptosisosteoarthritisimmunologydermatologyorthopedic surgerymembrane biologypharmacologylysosomal dysfunctioncartilage

Mitochondrial Dysfunction Triggers a Catabolic Response in Chondrocytes via Ros-Mediated Activation of the Jnk/Ap1 Pathway. Journal of Cell Science (2020)

mitochondrial dysfunctionmetabolismphysiologycell signalingosteoarthritiscell biologymetabolic pathwaysreactive oxygen speciescell metabolismredox signalingoxidative stressmitochondrial functionfree radical biologysignal transductioncatabolic responsejnk/ap1 pathwaycartilage

Oxidative Stress and Inflammation in Osteoarthritis Pathogenesis: Role of Polyphenols. Biomedicine & Pharmacotherapy (2020)

Mohammad Yunus AnsariNashrah AhmadTariq M Haqqi

inflammationrheumatologyosteoarthritismedicinepharmacologyoxidative stresspolyphenolicspharmacotherapyhealth sciencesosteoarthritis pathogenesis

Role of Inos in Osteoarthritis: Pathological and Therapeutic Aspects. Journal of Cellular Physiology (2020)

Nashrah AhmadMohammad Y. AnsariTariq M. Haqqi

cell physiologyinflammationpathologyregenerative medicinevascular biologyrheumatologyphysiologyosteoarthritiscellular physiologytissue physiologycellular therapybone diseasecellular biologymedicinetherapeutic aspectspain medicinestructural biology

Small Non-Coding Rnaome of Ageing Chondrocytes. International Journal of Molecular Sciences (2020)

Panagiotis BalaskasJonathan A. GreenTariq M. HaqqiPhilip DyerYalda A. KharazYongxiang FangXuan LiuTim J.M. WeltingMandy J. Peffers

small non-coding rnaomebiogerontologycell biologyageingmolecular biologygeneticsrna biologygenome biology

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Tariq Haqqi

Northeast Ohio Medical University

Affiliations

Department Of Cardiovascular Medicine

Northeast Ohio Medical University

NoneTariq Haqqi is currently serving as a Cardiovascular Medicine faculty at Northeast Ohio Medical University.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Tariq Haqqi

Northeast Ohio Medical University

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Erk1/2-Mediated Activation of Drp1 Regulates Mitochondrial Dynamics and Apoptosis in Chondrocytes. Osteoarthritis and Cartilage (2021)

M Y AnsariK NovakT M Haqqi

A Retrotransposon Gag-Like-3 Gene Rtl3 and Sox-9 Co-Regulate the Expression of Col2A1 in Chondrocytes. Connective Tissue Research (2020)

connective tissue diseasecell biologyneurosciencetissue physiologyhuman tissuestranscription (biology)geneticssignal transductiongene expressiontissue (biology)tissue engineeringbone research

Assessing Chondrocyte Status by Immunofluorescence-Mediated Localization of Parkin Relative to Mitochondria. Methods in molecular biology (2020)

Chondrocytes. Methods in molecular biology (2020)

Imperatorin Suppresses Il-1β-Induced Inos Expression via Inhibiting Erk-Mapk/Ap1 Signaling in Primary Human Oa Chondrocytes. International Immunopharmacology (2020)

Nashrah AhmadMohammad Y. AnsariShabana BanoTariq M Haqqi

immunologypharmacologydermatology

Lysosomal Dysfunction in Osteoarthritis and Aged Cartilage Triggers Apoptosis in Chondrocytes Through Bax Mediated Release of Cytochrome C. Osteoarthritis and Cartilage (2020)

M.Y. AnsariH.C. BallS.J. WaseK. NovakT.M. Haqqi

inflammationapoptosisosteoarthritisimmunologydermatologyorthopedic surgerymembrane biologypharmacologylysosomal dysfunctioncartilage

Mitochondrial Dysfunction Triggers a Catabolic Response in Chondrocytes via Ros-Mediated Activation of the Jnk/Ap1 Pathway. Journal of Cell Science (2020)

Oxidative Stress and Inflammation in Osteoarthritis Pathogenesis: Role of Polyphenols. Biomedicine & Pharmacotherapy (2020)

Mohammad Yunus AnsariNashrah AhmadTariq M Haqqi

inflammationrheumatologyosteoarthritismedicinepharmacologyoxidative stresspolyphenolicspharmacotherapyhealth sciencesosteoarthritis pathogenesis

Role of Inos in Osteoarthritis: Pathological and Therapeutic Aspects. Journal of Cellular Physiology (2020)

Nashrah AhmadMohammad Y. AnsariTariq M. Haqqi

Small Non-Coding Rnaome of Ageing Chondrocytes. International Journal of Molecular Sciences (2020)

Panagiotis BalaskasJonathan A. GreenTariq M. HaqqiPhilip DyerYalda A. KharazYongxiang FangXuan LiuTim J.M. WeltingMandy J. Peffers

small non-coding rnaomebiogerontologycell biologyageingmolecular biologygeneticsrna biologygenome biology

1
2
3
4
5
6
10 / page