Faculty

Institution

Unit

Venue

Sterling N. Wright, Ph.D.

Professor of Neurophysiology

Murray State University

Affiliations

Department Of Biology

Murray State University

Sterling N. Wright, Ph.d. is currently serving as a Biology Professor of Neurophysiology at Murray State University.

Honors

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Experiences

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Education

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Sterling N. Wright, Ph.D.

Murray State University

Professor of Neurophysiology

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electrophysiology physiology molecular pharmacology pharmacological research circulatory physiology biochemistry pharmacology neurophysiology molecular physiology neuropsychopharmacology neuropharmacology cellular physiology anesthesiology biophysical chemistry hypertension heart failure anesthesia metabolic pathways molecular metabolism cell metabolism cardiovascular research biological chemistry enzyme inhibition muscular dystrophy cellular biochemistry biochemical sciences protein science bioenergetics molecular psychiatry biophysical modeling steady-state inactivation na channel isoforms receptors drug metabolism bioorganic chemistry drug analysis neuroscience biosystems analytical toxicology bioanalysis nervous system addiction biology neural engineering membrane biology future medicinal chemistry drug discovery clinical chemistry molecular neuroscience chemical biology in vitro toxicology pharmacognosy neural circuits toxicology biophysics marine drug neurons central nervous system applied pharmacology spinal cord neuroanatomy toxins

Notable Scholarly Works

Notable Federal Grants

Collaborators

Modeling of Benzocaine Analog Interactions With the D4S6 Segment of Nav4.1 Voltage-Gated Sodium Channels. Biophysical Chemistry (2004)

chemical sciencesbiophysicsfuture medicinal chemistrychemical physicsmolecular modellingneuronselectrophysiologyneuropsychopharmacologymolecular sciencemolecular structuresmall moleculesbiochemistrychemistrymolecular pharmacologymolecular modelsbiophysical chemistrypharmacological research

Voltage-Dependent Inhibition of Rat Skeletal Muscle Sodium Channels by Aminoglycoside Antibiotics. Pflügers Archiv - European Journal of Physiology (2004)

antimicrobial agentscirculatory physiologyneuropharmacologyphysiologyelectrophysiologycellular physiologyneuropsychopharmacologyenzyme inhibitionbiochemistrydrug resistanceneurophysiologymolecular physiologypharmacologymolecular pharmacologyantibioticspharmacological researchreceptors

Cardiotoxic and Antiarrhythmic Tertiary Amine Local Anesthetics: Sodium Channel Affinity vs. Sodium Channel Gating. Current Vascular Pharmacology (2003)

cardiologycirculatory physiologyheart failureneuropharmacologyphysiologycardiovascular interventionsapplied pharmacologyanesthesiologyelectrophysiologyneuropsychopharmacologyneurophysiologypharmacologymolecular pharmacologyresuscitationpharmacological researchhypertensionanesthesia

Comparison of Aconitine‐Modified Human Heart (Hh1) and Rat Skeletal (μ1) Muscle Na + Channels: An Important Role for External Na + Ions. The Journal of Physiology (2002)

cardiovascular researchcirculatory physiologyheart failureanimal physiologyphysiologyanesthesiologyelectrophysiologycellular physiologymolecular physiologymuscular dystrophyneurophysiologypharmacologymolecular pharmacologypharmacological researchapplied physiologyhypertensioncardiovascular fitness

Irreversible Block of Human Heart (Hh1) Sodium Channels by the Plant Alkaloid Lappaconitine. Molecular Pharmacology (2001)

pharmacognosynatural product researchcardiologycirculatory physiologyheart failurenatural productsphysiologyelectrophysiologynatural product communicationmedicinal chemistrybiochemistrymarine drugpharmacologymolecular pharmacologypharmacological researchenzyme inhibitionhypertension

Coexpression With β 1 -Subunit Modifies the Kinetics and Fatty Acid Block of Hh1 α Na + Channels. American Journal of Physiology-Heart and Circulatory Physiology (2000)

biophysicsmetabolismenzymesmolecular metabolismphysiologyprotein sciencemetabolic pathwaysbiochemistrymolecular biophysicsbioenergeticscell metabolismmolecular physiologybiochemical sciencesmembrane biologybiological chemistryprotein foldingbiophysical chemistry

State-Dependent Cocaine Block of Sodium Channel Isoforms, Chimeras, and Channels Coexpressed With the β1 Subunit. Biophysical Journal (1999)

neuropharmacologyneuronsaddiction biologyneuropsychopharmacologyelectrophysiologyneuroscienceneurophysiologyaddictionnervous systemmolecular pharmacologypharmacological researchmolecular psychiatry

Fatty Acids Suppress Voltage-Gated Na + Currents in Hek293T Cells Transfected With the α-Subunit of the Human Cardiac Na + Channel. Proceedings of the National Academy of Sciences (1998)

lipidscardiovascular researchmetabolismcirculatory physiologymolecular metabolismphysiologycell signalingelectrophysiologycellular physiologymetabolic pathwayscell metabolismcell biologymolecular physiologymembrane biologybiochemistrysignal transductioncellular biochemistry

Lysine Point Mutations in Na+ Channel D4-S6 Reduce Inactivated Channel Block by Local Anesthetics. Molecular Pharmacology (1998)

molecular neurosciencecirculatory physiologyneuropharmacologyphysiologyanesthesiologyelectrophysiologycellular physiologyneuropsychopharmacologymolecular physiologyneurophysiologymuscular dystrophypharmacologybiological chemistrymolecular pharmacologypharmacological researchreceptorsanesthesia

Differences in Steady-State Inactivation Between Na Channel Isoforms Affect Local Anesthetic Binding Affinity. Biophysical Journal (1997)

S.N. WrightS.Y. WangR.G. KallenG.K. Wang

biophysicsna channel isoformssteady-state inactivationbiophysical modelingbiophysical chemistryanesthesia

1
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Sterling N. Wright, Ph.D.

Professor of Neurophysiology

Murray State University

Affiliations

Department Of Biology

Murray State University

Sterling N. Wright, Ph.d. is currently serving as a Biology Professor of Neurophysiology at Murray State University.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Sterling N. Wright, Ph.D.

Murray State University

Professor of Neurophysiology

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Modeling of Benzocaine Analog Interactions With the D4S6 Segment of Nav4.1 Voltage-Gated Sodium Channels. Biophysical Chemistry (2004)

Voltage-Dependent Inhibition of Rat Skeletal Muscle Sodium Channels by Aminoglycoside Antibiotics. Pflügers Archiv - European Journal of Physiology (2004)

Cardiotoxic and Antiarrhythmic Tertiary Amine Local Anesthetics: Sodium Channel Affinity vs. Sodium Channel Gating. Current Vascular Pharmacology (2003)

Comparison of Aconitine‐Modified Human Heart (Hh1) and Rat Skeletal (μ1) Muscle Na + Channels: An Important Role for External Na + Ions. The Journal of Physiology (2002)

Irreversible Block of Human Heart (Hh1) Sodium Channels by the Plant Alkaloid Lappaconitine. Molecular Pharmacology (2001)

Coexpression With β 1 -Subunit Modifies the Kinetics and Fatty Acid Block of Hh1 α Na + Channels. American Journal of Physiology-Heart and Circulatory Physiology (2000)

State-Dependent Cocaine Block of Sodium Channel Isoforms, Chimeras, and Channels Coexpressed With the β1 Subunit. Biophysical Journal (1999)

neuropharmacologyneuronsaddiction biologyneuropsychopharmacologyelectrophysiologyneuroscienceneurophysiologyaddictionnervous systemmolecular pharmacologypharmacological researchmolecular psychiatry

Fatty Acids Suppress Voltage-Gated Na + Currents in Hek293T Cells Transfected With the α-Subunit of the Human Cardiac Na + Channel. Proceedings of the National Academy of Sciences (1998)

Lysine Point Mutations in Na+ Channel D4-S6 Reduce Inactivated Channel Block by Local Anesthetics. Molecular Pharmacology (1998)

Differences in Steady-State Inactivation Between Na Channel Isoforms Affect Local Anesthetic Binding Affinity. Biophysical Journal (1997)

S.N. WrightS.Y. WangR.G. KallenG.K. Wang

biophysicsna channel isoformssteady-state inactivationbiophysical modelingbiophysical chemistryanesthesia

1
10 / page