Faculty

Institution

Unit

Venue

Stephen Moss

Professor

Tufts University

Affiliations

Graduate School of Biomedical Sciences

Neuroscience

School of Medicine

Neuroscience

Stephen Moss is currently serving as a Graduate School Of Biomedical Sciences Professor at Tufts University.

Honors

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Experiences

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Education

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Stephen Moss

Tufts University

Professor

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neuroscience neurology pharmacology neuropharmacology receptors molecular neuroscience molecular neurobiology neurochemistry neurophysiology molecular biology cell signaling brain science cellular neurobiology signal transduction systems neuroscience cognitive neuroscience protein science receptor (biochemistry) neural science biological chemistry neurotransmission molecular physiology protein phosphorylation neuropsychopharmacology molecular regulation gabaa receptors protein transport clinical neuroscience medicinal research mammalian biology protein function inhibitory synapses cell trafficking molecular psychiatry brain function psychopharmacology synaptic inhibition protein assembly functional regulation neurodynamics translational psychiatry redox biology biological psychiatry affective neuroscience pharmacotherapy cellular physiology intracellular trafficking gabab receptors camp-dependent protein kinase biochemical sciences animal physiology g protein-coupled receptor brain circuitry cell regulation functional modulation pharmacological research protein chemistry physiological research biomolecular interactions dopamine signaling pathways neuronal inhibition ligand-gated ion channels nucleus accumbens inhibitory synaptic transmission tyrosine phosphorylation subcellular localization protein kinases membrane insertion gaba receptors direct interaction functional modification neuroinflammation human physiology membrane transport host specificity

Notable Scholarly Works

Notable Federal Grants

Collaborators

Kcc2 Activation Reverses Neurophysiological and Behavioral Deficits in Female Rett Mice. bioRxiv (Cold Spring Harbor Laboratory) (2026)

female rett micebehavioral deficitsneuronsneurosciencepsychoneuroendocrinologybehavioral neurosciencephysiologyneurotransmissionneuroendocrine mechanismsneurophysiologyneuropsychopharmacology

Activation of Kcc2 During Development Alleviates Cognitive, Behavioral, and Neural Excitability in Adult Cdkl5-Deficient Mice. bioRxiv (Cold Spring Harbor Laboratory) (2025)

developmental biologydevelopment alleviatesneural excitabilitydevelopmental neurobiologyneuronsneuroscienceneurogeneticsmolecular neurobiologybehavioral neuroscienceneurotransmissiondevelopmental cognitive neuroscienceneurophysiologyadult cdkl5-deficient micecellular physiology

Kcc2 Inhibition and Neuronal Hyperexcitability Promote Extrinsic Apoptosis Dependent Upon C1q. Frontiers in Molecular Neuroscience (2025)

neuronal hyperexcitabilityapoptosisneuronscell biologycell signalingneuroprotectionneuroscienceextrinsic apoptosismolecular neurodegenerationkcc2 inhibitionneurophysiologycell deathcellular physiologyhyperpolarization (biology)

Neuroactive Steroids Activate Membrane Progesterone Receptors to Induce Sex Specific Effects on Protein Kinase Activity. iScience (2025)

membrane progesterone receptorsneuropharmacologyendocrinologyneurosciencesteroid metabolismneuroactive steroidsendocrine mechanismspharmacologyhormonal receptorsex researchreproductive biologyprotein kinase activitysexual medicinesexual and reproductive healthreproductive hormones

A Revised Nomenclature for the Lemur Family of Protein Kinases. Communications Biology (2024)

Gábor M. MórotzNeil A. BradburyOana CaluseriuShin-ichi HisanagaChristopher C. J. MillerAgnieszka Swiatecka-UrbanHeinz-Josef LenzStephen J. MossGeorgios Giamas

protein kinasesbiologylemur familymammalian biologyrevised nomenclaturecommunicationmolecular biology

Sustained Inhibition of Gaba-at by Ov329 Enhances Neuronal Inhibition and Prevents Development of Benzodiazepine Refractory Seizures. eNeuro (2024)

Phillip L. W. ColmersMuhammad Nauman ArshadJayanta MukherjeeShinghong LinShu Fun Josephine NgPatrick SarmierePaul A. DaviesStephen J. Moss

epilepsysystems neurosciencebenzodiazepine refractory seizuresneurosciencemolecular neurobiologypharmacologyprevents developmentneuropsychopharmacologyanesthetic pharmacologypsychotropic medicationneurologyneuropharmacologymolecular neuroscienceneuroprotectionneurodynamicsmedicinal researchcentral nervous systemmolecular psychiatrypain medicinepharmacotherapy

The Brain-Specific Kinase Lmtk3 Regulates Neuronal Excitability by Decreasing Kcc2-Dependent Neuronal CL− Extrusion. iScience (2024)

Noell ChoGeorgina KontouJoshua L SmalleyChristopher BopeJacob DenglerKristopher MontroseTarek Z DeebNicholas J BrandonTadashi YamamotoPaul A DaviesGeorgios GiamasStephen J Moss

brain mechanismsbrain circuitryneuronal excitabilityneurosciencecentral nervous systemneurodynamicsneurophysiologyneural circuitsbrain function

The Pde4 Inhibitor Apremilast Modulates Ethanol Responses in Gabrb1-S409A Knock-in Mice via Pka-Dependent and Independent Mechanisms. Neuropharmacology (2024)

Yuri A. BlednovWilliam ShawlotGregg E. HomanicsElizabeth A. Osterndorff-KahanekSonia MasonJody MayfieldJoshua L. SmalleyStephen J. MossRobert O. Messing

medicineneuropharmacologyneurosciencepharmacologymethanolindependent mechanisms

Direct Activation of Kcc2 Arrests Benzodiazepine Refractory Status Epilepticus and Limits the Subsequent Neuronal Injury in Mice. Cell Reports Medicine (2023)

Rebecca JarvisShu Fun Josephine NgAnna J. NathansonRoss A. CardarelliKrithika AbiramanFergus WadeAidan Evans-StrongMarina P. Fernandez-CampaTarek Z. DeebJoshua L. SmalleyTanguy JamierIan K. GurrellLisa McWilliamsAarti KawatkarLeslie C. ConwayQi WangRoland W. BurliNicholas J. BrandonIain P. ChessellAaron J. GoldmanJamie L. MaguireStephen J. Moss

epilepsyneuromodulation (medicine)medicinerefractory status epilepticusneuropharmacologycellular neurobiologyneuroprotectioncell signalingcentral nervous systempharmacologymammalian biologyneurotransmissionneurologycell detectionneurophysiologydirect activation

Estradiol Regulates Local Synthesis of Synaptic Proteome via Sex-Specific Mechanisms. bioRxiv (Cold Spring Harbor Laboratory) (2023)

Pooja RavalHannah RaeRodrigo R. R. DuarteIain A. WatsonKatherine J. SellersKathryn M. C. PachonLaura SichlingerTimothy R. PowellMarina V. YasvoinaJayanta MukherjeeStephen J. MossNicholas J. BrandonDeepak P. Srivastava

biophysicssystems neurosciencesex-specific mechanismsneurosciencesynaptic proteomeneurologylocal synthesis

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Stephen Moss

Professor

Tufts University

Affiliations

Graduate School of Biomedical Sciences

Neuroscience

School of Medicine

Neuroscience

Stephen Moss is currently serving as a Graduate School Of Biomedical Sciences Professor at Tufts University.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Stephen Moss

Tufts University

Professor

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Kcc2 Activation Reverses Neurophysiological and Behavioral Deficits in Female Rett Mice. bioRxiv (Cold Spring Harbor Laboratory) (2026)

female rett micebehavioral deficitsneuronsneurosciencepsychoneuroendocrinologybehavioral neurosciencephysiologyneurotransmissionneuroendocrine mechanismsneurophysiologyneuropsychopharmacology

Activation of Kcc2 During Development Alleviates Cognitive, Behavioral, and Neural Excitability in Adult Cdkl5-Deficient Mice. bioRxiv (Cold Spring Harbor Laboratory) (2025)

Kcc2 Inhibition and Neuronal Hyperexcitability Promote Extrinsic Apoptosis Dependent Upon C1q. Frontiers in Molecular Neuroscience (2025)

Neuroactive Steroids Activate Membrane Progesterone Receptors to Induce Sex Specific Effects on Protein Kinase Activity. iScience (2025)

A Revised Nomenclature for the Lemur Family of Protein Kinases. Communications Biology (2024)

Gábor M. MórotzNeil A. BradburyOana CaluseriuShin-ichi HisanagaChristopher C. J. MillerAgnieszka Swiatecka-UrbanHeinz-Josef LenzStephen J. MossGeorgios Giamas

protein kinasesbiologylemur familymammalian biologyrevised nomenclaturecommunicationmolecular biology

Sustained Inhibition of Gaba-at by Ov329 Enhances Neuronal Inhibition and Prevents Development of Benzodiazepine Refractory Seizures. eNeuro (2024)

Phillip L. W. ColmersMuhammad Nauman ArshadJayanta MukherjeeShinghong LinShu Fun Josephine NgPatrick SarmierePaul A. DaviesStephen J. Moss

The Brain-Specific Kinase Lmtk3 Regulates Neuronal Excitability by Decreasing Kcc2-Dependent Neuronal CL− Extrusion. iScience (2024)

Noell ChoGeorgina KontouJoshua L SmalleyChristopher BopeJacob DenglerKristopher MontroseTarek Z DeebNicholas J BrandonTadashi YamamotoPaul A DaviesGeorgios GiamasStephen J Moss

brain mechanismsbrain circuitryneuronal excitabilityneurosciencecentral nervous systemneurodynamicsneurophysiologyneural circuitsbrain function

The Pde4 Inhibitor Apremilast Modulates Ethanol Responses in Gabrb1-S409A Knock-in Mice via Pka-Dependent and Independent Mechanisms. Neuropharmacology (2024)

Yuri A. BlednovWilliam ShawlotGregg E. HomanicsElizabeth A. Osterndorff-KahanekSonia MasonJody MayfieldJoshua L. SmalleyStephen J. MossRobert O. Messing

medicineneuropharmacologyneurosciencepharmacologymethanolindependent mechanisms

Direct Activation of Kcc2 Arrests Benzodiazepine Refractory Status Epilepticus and Limits the Subsequent Neuronal Injury in Mice. Cell Reports Medicine (2023)

Estradiol Regulates Local Synthesis of Synaptic Proteome via Sex-Specific Mechanisms. bioRxiv (Cold Spring Harbor Laboratory) (2023)

biophysicssystems neurosciencesex-specific mechanismsneurosciencesynaptic proteomeneurologylocal synthesis

1
2
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5
26
10 / page

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