Faculty

Institution

Unit

Venue

Stephen A. Back

Oregon Health & Science University

Affiliations

OHSU School of Medicine

Department Of Pediatrics

Division Of Neurology

Oregon Health & Science University

NoneStephen A. Back is currently serving as a Pediatrics faculty at Oregon Health & Science University.

Honors

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Experiences

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Education

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Stephen A. Back

Oregon Health & Science University

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neuroscience neurology developmental neurobiology brain injury developmental biology neuropathology white matter neurophysiology neonatology child neurology central nervous system pediatrics neuroradiology molecular neurodegeneration cerebral blood flow neurodevelopmental disorders preterm birth cellular physiology neuroanatomy brain science developmental cognitive neuroscience hypoxia (medicine) maternal health neuroimaging pregnancy hypertension animal physiology matrix biology neuroregeneration neurorehabilitation vascular biology neural science clinical neurology brain lesion molecular neurobiology mammalian biology obstetrics and gynaecology neuroinflammation neural stem cells brain structure cellular neurobiology cell death free radical biology maternal-fetal medicine clinical neuroscience brain circulation geriatric neurology neurochemistry ischemic syndrome experimental neuropathology placental development white matter injury neurotology early life exposures apoptosis brain imaging tissue injury neuroprotection theriogenology brain pathology brain-immune interaction intracranial pressure selective vulnerability preterm infant oligodendrocyte lineage maturation neonatal hypoxia-ischemia isoflurane-induced apoptosis cerebral palsy premature infant transient hypoxemia oligodendrocyte progenitors developmental window late oligodendrocyte progenitors demyelinated lesions premature infants preoligodendrocyte maturation contributes premature neonates

Notable Scholarly Works

Notable Federal Grants

Collaborators

Mild Neonatal Hypoxia Targets Synaptic Maturation, Disrupts Adult Hippocampal Learning and Memory, and is Associated With Ck2-Mediated Loss of Synaptic Calcium-Activated Potassium Channel Kcnn2 Activity. Journal of Neuroscience (2026)

ck2-mediated losshippocampal learningmolecular neuroscienceneuronsneurosciencemolecular neurobiologyphysiologymolecular neurodegenerationneurotransmissionneurophysiologyneural circuitscellular physiology

Commentary on: “Delayed Necrostatin-1S Infusion Attenuates Cystic White Matter Injury in Preterm Fetal Sheep.”. Neurotherapeutics (2025)

developmental biologyplacental developmentanimal healthpreterm fetal sheepveterinary medicineveterinary scienceanimal scienceanimal physiologycaprinephysiologyembryologyclinical research

Hyaluronidases as Targets for the Treatment of Neurological Diseases. Proteoglycan Research (2025)

neuropathologywound healingneurodegenerative diseasesneuro-ophthalmologymatrix biologyneural tissue engineeringmolecular neurodegenerationneurological diseasesneurology

The Cemip Hyaluronidase is Elevated in Oligodendrocyte Progenitor Cells and Inhibits Oligodendrocyte Maturation. ASN NEURO (2025)

developmental biologystem cell researchcell specializationconnective tissue diseasecellular differentiationcemip hyaluronidasehuman tissuesstem cellscell biologyoligodendrocyte progenitor cellsdevelopmental cellinhibits oligodendrocyte maturationextracellular matrixmatrix biologytissue (biology)cellular physiology

Contributors. Elsevier eBooks (2024)

Distinct Chemical Structures Inhibit the Cemip Hyaluronidase and Promote Oligodendrocyte Progenitor Cell Maturation. Journal of Biological Chemistry (2024)

developmental biologystem cell researchcell specializationcellular differentiationcemip hyaluronidasecell biologydevelopmental cellcell-matrix interactionsextracellular matrixmatrix biologycell and molecular biologydistinct chemical structurescellular physiologycellular biochemistry

Encephalopathy of Prematurity: Pathophysiology. Elsevier eBooks (2024)

epilepsyneuropathologyneurological disordersmaternal healthdevelopmental toxicologyneuroscienceearly life exposurespregnancy hypertensionchild neurologyneurologyneurophysiology

Mild Neonatal Hypoxia Disrupts Adult Hippocampal Learning and Memory and is Associated With Ck2-Mediated Dysregulation of Synaptic Calcium-Activated Potassium Channel Kcnn2. bioRxiv (Cold Spring Harbor Laboratory) (2024)

hippocampal learningmolecular neuroscienceneuronsneuroscienceneurogeneticsmolecular neurobiologyck2-mediated dysregulationphysiologymolecular neurodegenerationneurotransmissionneurophysiologyneural circuitscellular physiologymolecular physiologyhyperpolarization (biology)

Tsg-6–Mediated Extracellular Matrix Modifications Regulate Hypoxic–Ischemic Brain Injury. Journal of Neuroscience (2024)

Taasin SrivastavaHung NguyenGage HadenParham DibaSteven SowaNorah LaNguyenWilliam Reed-DustinWenbin ZhuXi GongEdward N. HarrisSelva BaltanStephen A. Back

systems neuroscienceneurochemistrycellular neurobiologymolecular neuroscienceclinical neurosciencehypoxic-ischemic brain injurybrain scienceprotein scienceneuroscienceextracellular matrix modificationsextracellular matrixneurologybrain injurynervous systemhost specificity

Brain Injury in the Preterm Infant. Avery's Diseases of the Newborn (2023)

traumaneuropathologypediatric neurosurgerypreterm birthneonatologypediatricschild neurologyneurologydevelopmental cognitive neurosciencebrain injury

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Stephen A. Back

Oregon Health & Science University

Affiliations

OHSU School of Medicine

Department Of Pediatrics

Division Of Neurology

Oregon Health & Science University

NoneStephen A. Back is currently serving as a Pediatrics faculty at Oregon Health & Science University.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Stephen A. Back

Oregon Health & Science University

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Commentary on: “Delayed Necrostatin-1S Infusion Attenuates Cystic White Matter Injury in Preterm Fetal Sheep.”. Neurotherapeutics (2025)

developmental biologyplacental developmentanimal healthpreterm fetal sheepveterinary medicineveterinary scienceanimal scienceanimal physiologycaprinephysiologyembryologyclinical research

Hyaluronidases as Targets for the Treatment of Neurological Diseases. Proteoglycan Research (2025)

neuropathologywound healingneurodegenerative diseasesneuro-ophthalmologymatrix biologyneural tissue engineeringmolecular neurodegenerationneurological diseasesneurology

The Cemip Hyaluronidase is Elevated in Oligodendrocyte Progenitor Cells and Inhibits Oligodendrocyte Maturation. ASN NEURO (2025)

Contributors. Elsevier eBooks (2024)

Distinct Chemical Structures Inhibit the Cemip Hyaluronidase and Promote Oligodendrocyte Progenitor Cell Maturation. Journal of Biological Chemistry (2024)

Encephalopathy of Prematurity: Pathophysiology. Elsevier eBooks (2024)

epilepsyneuropathologyneurological disordersmaternal healthdevelopmental toxicologyneuroscienceearly life exposurespregnancy hypertensionchild neurologyneurologyneurophysiology

Tsg-6–Mediated Extracellular Matrix Modifications Regulate Hypoxic–Ischemic Brain Injury. Journal of Neuroscience (2024)

Taasin SrivastavaHung NguyenGage HadenParham DibaSteven SowaNorah LaNguyenWilliam Reed-DustinWenbin ZhuXi GongEdward N. HarrisSelva BaltanStephen A. Back

Brain Injury in the Preterm Infant. Avery's Diseases of the Newborn (2023)

traumaneuropathologypediatric neurosurgerypreterm birthneonatologypediatricschild neurologyneurologydevelopmental cognitive neurosciencebrain injury

1
2
3
4
5
9
10 / page