M Saeed Sheikh

Upstate Medical University

Affiliations

Norton College of Medicine

Department Of Anesthesiology

Department Of Pharmacology

Institute Of Suny Upstate Cancer Research

Department Of Anesthesiology

Norton College Of Medicine

Upstate Medical University

NoneM Saeed Sheikh is currently serving as a Anesthesiology faculty at Upstate Medical University.

Honors

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Experiences

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Education

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M Saeed Sheikh

Upstate Medical University

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molecular biology tumor biology cell biology oncogenes cell death cancer cells cancer research cancer biology apoptosis medicinal research systems biology prostate cancer tumor microenvironment therapeutic targets molecular cancer biology carcinogenesis oncology cancer immunotherapy cellular therapy vascular biology enzyme inhibition cell metabolism cellular biology colorectal cancer cancer treatment tumor suppressors anti-cancer agents monoglyceride lipase cancer cell sensitivity dna damage human malignancies nongenotoxic stress-regulated gene brain tumor research surgical oncology molecular neurodegeneration cellular physiology experimental medicine cancer care cancer discovery inhibitory activity thermally activated delayed fluorescence treatment and prevention in vivo systems localized excited state cell physiology cancer genetics cellular senescence gastrointestinal oncology viral oncology protein transport t-regulatory cell protein function cancer management drug targeting oncogenic agent g protein-coupled receptor disease progression cell proliferation tumor heterogeneity cell cycle gene characterization protein expression lipoprotein metabolism cell regulation uniform nomenclature cristae organizing system mitochondrial contact site decoy receptors microtubule-binding protein death receptors human colorectal cancer potential tumor-suppressive role rna-binding protein hur negative regulation lung adenocarcinoma

Notable Scholarly Works

Notable Federal Grants

Collaborators

Cell Death, Molecular Targeted Therapies, and Metabolic Reprogramming in Egfr-Mutant Lung Cancer. Cancers (2025)

High Glucose and Glucose-Derived Intermediates Are Linked to Lung Cancer Aggressiveness. Current Cancer Drug Targets (2025)

Metabolic Stress–Mediated Cell Death and Autophagy in Human Lung Cancer Cells. Journal of Pharmacology and Experimental Therapeutics (2025)

Ecrg2/Spink7 Tumor Suppressor as Modulator of Dna Damage Response. International Journal of Molecular Sciences (2024)

Data From Lappaol F, a Novel Anticancer Agent Isolated From Plant I Arctium Lappa /I L.. (2023)

Sacituzumab Govitecan for Hormone Receptor-Positive and Triple-Negative Breast Cancers.. PubMed (2023)

Supplementary Data From Doc45, a Novel Dna Damage–Regulated Nucleocytoplasmic Atpase That is Overexpressed in Multiple Human Malignancies. (2023)

Supplementary Figures 1 Through 7 From Lappaol F, a Novel Anticancer Agent Isolated From Plant I Arctium Lappa /I L.. (2023)

Supplementary Tables 1-3 From Cloning and Characterization of a P53 and Dna Damage Down-Regulated Gene I Piq /I That Codes for a Novel Calmodulin-Binding Iq Motif Protein and is Up-Regulated in Gastrointestinal Cancers. (2023)

Supplementary Tables S1-S4 From I Neutral Sphingomyelinase-3 /I is a Dna Damage and Nongenotoxic Stress-Regulated Gene That is Deregulated in Human Malignancies. (2023)

Chad A. CorcoranQin HeSuriyan PonnusamyBesim OgretmenYing HuangM. Saeed Sheikh

nongenotoxic stress-regulated genedna damagehuman malignanciesmolecular biologydna repair

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