Faculty

Institution

Unit

Venue

Robert Marr

Rosalind Franklin University of Medicine and Science

Affiliations

Chicago Medical School

Center for Neurodegenerative Disease and Therapeutics

Foundational Sciences and Humanities

Midwest Proteome Center

School Of Chicago Medical

College of Health Professions

Department Of Physician Assistant

School Of Chicago Medical

Department Of Clinical Sciences

Department Of Foundational Sciences And Humanities

Foundational Sciences and Humanities

Department of Humanities & Health Care

Illinois Board of Higher Education

Laboratories Of Kastor Neurobiology Of Aging

Rosalind Franklin University of Medicine and Science

NoneRobert Marr is currently serving as a Center For Neurodegenerative Disease And Therapeutics faculty at Rosalind Franklin University of Medicine And Science.

Honors

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Experiences

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Education

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Robert Marr

Rosalind Franklin University of Medicine and Science

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neuroscience alzheimer's disease molecular neurodegeneration neurology neurodegenerative diseases molecular neuroscience molecular neurobiology neurogenetics genetics neuroscience of aging brain science cognitive neuroscience cognitive impairment developmental neurobiology degenerative pathology pharmacology biological psychiatry protein science enzyme inhibition mammalian biology molecular psychiatry neuropsychopharmacology medicinal research clinical neuroscience brain function neuroprotection stress (biology) psychiatric research aging cell affective neuroscience stem cell research head trauma rehabilitation experimental gerontology experimental neuropathology splicing variant neural science protein misfolding memory loss brain pathology neuroregeneration gene transfer protein function transgenic model mild cognitive impairment psychoneuroendocrinology biological psychology behavioral neuroendocrinology experimental psychopathology psychoneuroimmunology neurochemistry neurobiological mechanisms neuroimaging biomarkers nucleic acid amplification oligonucleotides host specificity aging-associated diseases molecular pathology disease progression protein transport functional regulation antisense therapy stress biomarkers regulate anxiety disease neuropathology sirnas ameliorates human amyloid pathology neprilysin gene transfer transgenic mice ryanodine receptor expression antisense oligonucleotides therapeutic correction neprilysin processing amyloid-beta clearance adult brain calcium dysregulation protein mishandling

Notable Scholarly Works

Notable Federal Grants

Collaborators

CORRECTION: Peripheral Delivery of a Cns Targeted, Metalo-Protease Reduces Aβ Toxicity in a Mouse Model of Alzheimer’s Disease. PLoS ONE (2025)

Long-Term Neprilysin Gene Transfer is Associated With Reduced Levels of Intracellular Abeta and Behavioral Improvement in App Transgenic Mice. BMC Neuroscience (2025)

receptorsmolecular cell biologymolecular psychiatrygene expressionneurogeneticsgene therapymolecular medicinemolecular physiologymolecular neurodegenerationdevelopmental biologydevelopmental cellneuropsychopharmacologymolecular neurosciencedevelopmental neurobiologycell biologymammalian biologyphysiology

Mitochondrial Dysfunction Mediated by Er-Calcium Dysregulation in Neurons Derived From Alzheimer’s Disease Patients. Acta Neuropathologica Communications (2025)

neurologyalzheimer's diseasemitochondrial functioncognitive impairmentmitochondrial dysfunctionneurosciencecell metabolismmolecular neurodegenerationfree radical biologyer-calcium dysregulationneurodegenerative diseasesdegenerative pathology

Neprilysin-2. Handbook of Proteolytic Enzymes (2025)

Mitochondrial Dysfunction in Alzheimer’s Disease is Driven by Excess Er-Calcium Release in Patient-Derived Neurons. bioRxiv (Cold Spring Harbor Laboratory) (2024)

Sarah Mustaly-KalimiWacey GallegosRobert A. MarrDaniel A. PetersonIsrael SeklerGrace E. Stutzmann

alzheimer's diseasemitochondrial dysfunctionneuroscienceexcess er-calcium releaseneurodegenerative diseasespatient-derived neurons

Engineered Neurogenesis in NaïVe Adult Rat Cortex by Ngn2-Mediated Neuronal Reprogramming of Resident Oligodendrocyte Progenitor Cells. Frontiers in Neuroscience (2023)

Stanley F. BazarekMentor ThaqiPatrick KingAmol R. MehtaRonil PatelClark A. BriggsEmily ReisenbiglerJonathon E. YouseyElis A. MillerGrace E. StutzmannRobert A. MarrDaniel A. Peterson

neuroanatomyneuroscience

Protein Mishandling and Impaired Lysosomal Proteolysis Generated Through Calcium Dysregulation in Alzheimer’s Disease. Proceedings of the National Academy of Sciences (2022)

Sarah Mustaly-KalimiWacey GallegosRobert A. MarrAlice Gilman-SachsDaniel A. PetersonIsrael SeklerGrace E. Stutzmann

impaired lysosomal proteolysisfunctional regulationneurodegenerative diseasesprotein sciencealzheimer's diseasemembrane biologyproteomicsneurosciencemolecular neurodegenerationvascular biologyneurologyinflammationprotein mishandlingmammalian biologydisease progressioncalcium dysregulationprotein functionmedicinal researchprotein misfoldingprotein transportphysiology

Induced Neurons for Disease Modeling and Repair: A Focus on Non-Fibroblastic Cell Sources in Direct Reprogramming. Frontiers in Bioengineering and Biotechnology (2021)

Kathryn M. KimMentor ThaqiDaniel A. PetersonRobert A. Marr

disease modelingdirect reprogrammingnon-fibroblastic cell sourcesneuroscienceinduced neuronsbiotechnologybiomedical engineeringbiological engineering

Neuropathological Signaling in Ad Human Induced Neurons. Alzheimer s & Dementia (2021)

neuroscienceneuronsmolecular neurodegenerationmolecular neurobiologyad humanneurodegenerative diseases

Assessment of the Effects of Altered Amyloid-Beta Clearance on Behavior Following Repeat Closed-Head Brain Injury in Amyloid-Beta Precursor Protein Humanized Mice. Journal of Neurotrauma (2020)

Kathleen C. MaiglerTrevor J. BuhrChristopher S. ParkSteven A. MillerDorothy A. KozlowskiRobert A. Marr

neuropathologybrain pathologyneurodegenerative diseasesneuroinflammationbehavioral neuroscienceamyloid-beta precursor proteinneurosciencemolecular neurodegenerationneurologyaltered amyloid-beta clearancehead trauma rehabilitationmammalian biologyneurorehabilitationbrain injurybrain sciencebrain functionexperimental neuropathologycognitive neurosciencebehavioral sciences

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Robert Marr

Rosalind Franklin University of Medicine and Science

Affiliations

Chicago Medical School

Center for Neurodegenerative Disease and Therapeutics

Foundational Sciences and Humanities

Midwest Proteome Center

School Of Chicago Medical

College of Health Professions

Department Of Physician Assistant

School Of Chicago Medical

Department Of Clinical Sciences

Department Of Foundational Sciences And Humanities

Foundational Sciences and Humanities

Department of Humanities & Health Care

Illinois Board of Higher Education

Laboratories Of Kastor Neurobiology Of Aging

Rosalind Franklin University of Medicine and Science

NoneRobert Marr is currently serving as a Center For Neurodegenerative Disease And Therapeutics faculty at Rosalind Franklin University of Medicine And Science.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Robert Marr

Rosalind Franklin University of Medicine and Science

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

CORRECTION: Peripheral Delivery of a Cns Targeted, Metalo-Protease Reduces Aβ Toxicity in a Mouse Model of Alzheimer’s Disease. PLoS ONE (2025)

Long-Term Neprilysin Gene Transfer is Associated With Reduced Levels of Intracellular Abeta and Behavioral Improvement in App Transgenic Mice. BMC Neuroscience (2025)

Mitochondrial Dysfunction Mediated by Er-Calcium Dysregulation in Neurons Derived From Alzheimer’s Disease Patients. Acta Neuropathologica Communications (2025)

Neprilysin-2. Handbook of Proteolytic Enzymes (2025)

Mitochondrial Dysfunction in Alzheimer’s Disease is Driven by Excess Er-Calcium Release in Patient-Derived Neurons. bioRxiv (Cold Spring Harbor Laboratory) (2024)

Sarah Mustaly-KalimiWacey GallegosRobert A. MarrDaniel A. PetersonIsrael SeklerGrace E. Stutzmann

alzheimer's diseasemitochondrial dysfunctionneuroscienceexcess er-calcium releaseneurodegenerative diseasespatient-derived neurons

Engineered Neurogenesis in NaïVe Adult Rat Cortex by Ngn2-Mediated Neuronal Reprogramming of Resident Oligodendrocyte Progenitor Cells. Frontiers in Neuroscience (2023)

Stanley F. BazarekMentor ThaqiPatrick KingAmol R. MehtaRonil PatelClark A. BriggsEmily ReisenbiglerJonathon E. YouseyElis A. MillerGrace E. StutzmannRobert A. MarrDaniel A. Peterson

neuroanatomyneuroscience

Protein Mishandling and Impaired Lysosomal Proteolysis Generated Through Calcium Dysregulation in Alzheimer’s Disease. Proceedings of the National Academy of Sciences (2022)

Sarah Mustaly-KalimiWacey GallegosRobert A. MarrAlice Gilman-SachsDaniel A. PetersonIsrael SeklerGrace E. Stutzmann

Induced Neurons for Disease Modeling and Repair: A Focus on Non-Fibroblastic Cell Sources in Direct Reprogramming. Frontiers in Bioengineering and Biotechnology (2021)

Kathryn M. KimMentor ThaqiDaniel A. PetersonRobert A. Marr

disease modelingdirect reprogrammingnon-fibroblastic cell sourcesneuroscienceinduced neuronsbiotechnologybiomedical engineeringbiological engineering

Neuropathological Signaling in Ad Human Induced Neurons. Alzheimer s & Dementia (2021)

neuroscienceneuronsmolecular neurodegenerationmolecular neurobiologyad humanneurodegenerative diseases

Assessment of the Effects of Altered Amyloid-Beta Clearance on Behavior Following Repeat Closed-Head Brain Injury in Amyloid-Beta Precursor Protein Humanized Mice. Journal of Neurotrauma (2020)

Kathleen C. MaiglerTrevor J. BuhrChristopher S. ParkSteven A. MillerDorothy A. KozlowskiRobert A. Marr

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10 / page