Faculty

Institution

Unit

Venue

Michael P. Davey, M.D., Ph.D.

Professor of Medicine

Oregon Health & Science University

Affiliations

OHSU School of Medicine

Department Of Molecular Microbiology And Immunology

Michael P. Davey, M.d., Ph.d. is currently serving as a Molecular Microbiology And Immunology Professor of Medicine at Oregon Health & Science University.

Honors

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Experiences

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Education

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Michael P. Davey, M.D., Ph.D.

Oregon Health & Science University

Professor of Medicine

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immunology immune responses inflammation cell biology arthritis genetics dermatology autoimmunity cell signaling hematology cellular therapy medicinal research cellular immune responses clinical immunology cytokines rheumatology autoimmune diseases protein science clinical rheumatology ophthalmology retina ocular disease experimental ophthalmology biophysical chemistry reproductive biology animal physiology biological chemistry mammalian biology reproductive immunology autophagy stem cell research liver transplantation liver physiology oral biology reproductive physiology protein function animal reproduction protein transport mammalian physiology inhibitory activity residual stress immunological memory psychoneuroimmunology disease progression inflammatory rheumatic disease molecular regulation inflammatory diseases adaptive immune system innate immune system t-regulatory cell ocular pathology chronic inflammation immune dysregulation autoantibody production peptide engineering peptide synthesis peptide therapeutics neuropeptides protein chemistry granulocyte immunological biomarkers mammalian sperm motility selective depletion autoimmune encephalomyelitis blau syndrome mutation a-kinase anchoring proteins autoimmune uveitis ocular inflammation mouse model familial granulomatous uveitis exacerbates autoimmune arthritis murine model proteoglycan-induced arthritis riia regulatory subunit murine immune system rapid analysis novel model

Notable Scholarly Works

Notable Federal Grants

Collaborators

Nod2-Deficient Cd4+ T Cells Protect Against Candida Albicans Infection. The Journal of Immunology (2021)

T Cell-Intrinsic Role for Nod2 in Protection Against Th17-Mediated Uveitis. Nature Communications (2020)

Ruth J. NapierEllen J. LeeMichael P. DaveyEmily E. VanceJoão M. FurtadoPaige E. SnowKimberly A. SamsonSydney J. LashleyBrieanna R. BrownReiko HoraiMary J. MattapallilBiying XuMichelle C. CalleganLuke S. UebelhoerChristina L. LancioniRichard K. VeheBryce A. BinstadtJustine R. SmithRachel R. CaspiHolly L. Rosenzweig

t-regulatory cellcell signalingimmunologymedicinal researchdermatologycellular therapyimmune responsescellular immune responsespharmacologyadaptive immune systeminnate immune systemcytokines

Nod2 Deficiency Augments Th17 Responses and Exacerbates Autoimmune Arthritis. The Journal of Immunology (2018)

inflammationexacerbates autoimmune arthritisarthritisimmunologyimmune dysregulationimmune responsesautoantibody productionautoimmune diseasesautoimmunityclinical rheumatology

Nod2 is a Negative Regulator of Arthritogenic T Cell Responses in Skg Mice. The Journal of Immunology (2018)

Blau Syndrome–Associated Nod2 Mutation Alters Expression of Full-Length Nod2 and Limits Responses to Muramyl Dipeptide in Knock-in Mice. The Journal of Immunology (2014)

Nucleotide-Binding and Oligomerization Domain 2 (Nod2) Knock-in Mice Carrying a Mutation Associated With Blau Syndrome Show Reduced Amounts of Nod2 Protein and Decreased Muramyl Dipeptide (Mdp)-Induced Inflammatory Responses (P1254). The Journal of Immunology (2013)

Dectin-1 and Nod2 Mediate Cathepsin Activation in Zymosan-Induced Arthritis in Mice. Inflammation Research (2011)

Interferon‐γ Regulates Discordant Mechanisms of Uveitis Versus Joint and Axial Disease in a Murine Model Resembling Spondylarthritis. Arthritis & Rheumatism (2011)

Nod2 Deficiency Results in Increased Susceptibility to Peptidoglycan-Induced Uveitis in Mice. Investigative Ophthalmology & Visual Science (2011)

Reply. Arthritis & Rheumatism (2011)

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Michael P. Davey, M.D., Ph.D.

Professor of Medicine

Oregon Health & Science University

Affiliations

OHSU School of Medicine

Department Of Molecular Microbiology And Immunology

Michael P. Davey, M.d., Ph.d. is currently serving as a Molecular Microbiology And Immunology Professor of Medicine at Oregon Health & Science University.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Michael P. Davey, M.D., Ph.D.

Oregon Health & Science University

Professor of Medicine

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Nod2-Deficient Cd4+ T Cells Protect Against Candida Albicans Infection. The Journal of Immunology (2021)

T Cell-Intrinsic Role for Nod2 in Protection Against Th17-Mediated Uveitis. Nature Communications (2020)

t-regulatory cellcell signalingimmunologymedicinal researchdermatologycellular therapyimmune responsescellular immune responsespharmacologyadaptive immune systeminnate immune systemcytokines

Nod2 Deficiency Augments Th17 Responses and Exacerbates Autoimmune Arthritis. The Journal of Immunology (2018)

inflammationexacerbates autoimmune arthritisarthritisimmunologyimmune dysregulationimmune responsesautoantibody productionautoimmune diseasesautoimmunityclinical rheumatology

Nod2 is a Negative Regulator of Arthritogenic T Cell Responses in Skg Mice. The Journal of Immunology (2018)

Blau Syndrome–Associated Nod2 Mutation Alters Expression of Full-Length Nod2 and Limits Responses to Muramyl Dipeptide in Knock-in Mice. The Journal of Immunology (2014)

Dectin-1 and Nod2 Mediate Cathepsin Activation in Zymosan-Induced Arthritis in Mice. Inflammation Research (2011)

Interferon‐γ Regulates Discordant Mechanisms of Uveitis Versus Joint and Axial Disease in a Murine Model Resembling Spondylarthritis. Arthritis & Rheumatism (2011)

Nod2 Deficiency Results in Increased Susceptibility to Peptidoglycan-Induced Uveitis in Mice. Investigative Ophthalmology & Visual Science (2011)

Reply. Arthritis & Rheumatism (2011)

1
2
3
4
5
6
10 / page