Faculty

Institution

Unit

Venue

Menashe Bar-Eli

The University of Texas MD Anderson Cancer Center

Affiliations

Center For Rna Interference And Non-Coding Rna

Department Of Cancer Biology

MD Anderson UTHealth Houston Graduate School

The University of Texas MD Anderson Cancer Center

NoneMenashe Bar-eli is currently serving as a Center For Rna Interference And Non-Coding Rna faculty at The University of Texas Md Anderson Cancer Center.

Honors

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Experiences

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Education

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Menashe Bar-Eli

The University of Texas MD Anderson Cancer Center

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tumor biology cancer cells cell biology melanoma cancer biology oncogenes cancer research metastasis molecular cancer oncology immunology pathology molecular biology medicinal research skin cancer cell signaling carcinogenesis cancer growth gene expression cancer immunotherapy vascular biology molecular cell biology tumor suppressors cancer genetics leukemia cellular therapy cancer genomics cell regulation cancer treatment cancer discovery photocarcinogenesis tumor targeting signaling pathways melanoma metastasis tumor growth cancer care cytokines genitourinary cancers human melanoma anti-cancer agents human melanoma cells melanoma progression cell death molecular diagnostics receptor tyrosine kinases angiogenesis cancer immunosurveillance biological chemistry gynecologic oncology brain tumor research cell adhesion microrna detection colorectal cancer melanoma cells gene regulation immune checkpoint inhibitors molecular regulation apoptosis in vivo systems tumor heterogeneity tumor progression medical mycology rna biology breast oncology immunomodulation clinical pathology human bladder cancer prostatic disease expression regulates tumorigenicity interleukin 8 metastatic melanoma src activation key switch melanoma growth human antibodies thrombin receptor

Notable Scholarly Works

Notable Federal Grants

Collaborators

Abstract 5073: Nt219 Induces Tumor Pd-L1 Expression and Potentiates Anti-Pd-1 Efficacy. Cancer Research (2023)

Ricardo Alexandre de AzevedoHadas ReuveniMenashe Bar-EliMichael Curran

tumor biologygene expressiononcologycancer researchcancer biologymolecular biologytumor microenvironment

Conflict of Interest Form 1 From Mir-200 Expression Regulates Epithelial-To-Mesenchymal Transition in Bladder Cancer Cells and Reverses Resistance to Epidermal Growth Factor Receptor Therapy. (2023)

bladder cancer cellsurologytumor biologymolecular cancercancer biologycell biologycancer cellsmolecular cell biologyoncogenesreverses resistancebladder cancersmall rnainterest form 1

Data From a Subset of Host B Lymphocytes Controls Melanoma Metastasis Through a Melanoma Cell Adhesion Molecule/Muc18-Dependent Interaction: Evidence From Mice and Humans. (2023)

metastasisimmunomodulationtumor biologyimmune responsescell biologycancer cellsa subsetcancer immunosurveillanceimmunologycell researchmelanomatumor microenvironment

Data From Expression of Id-1 is Regulated by Mcam/Muc18: A Missing Link in Melanoma Progression. (2023)

Maya ZiglerGabriel J. VillaresAndrey S. DobroffHua WangLi HuangRussell R. BraeuerTakafumi KamiyaVladislava O. MelnikovaRenduo SongRan FriedmanRhoda M. AlaniMenashe Bar-Eli

tumor biologygeneticsmelanoma progressionmelanomacancer researchoncologycancer biologydermatologydata sciencegene expressionskin cancercancer genetics

Data From Melanoma Proteoglycan Modifies Gene Expression to Stimulate Tumor Cell Motility, Growth, and Epithelial-To-Mesenchymal Transition. (2023)

Jianbo YangMatthew A. PriceGui Yuan LiMenashe Bar-EliRavi SalgiaRamasamy JagedeeswaranJennifer H. CarlsonSoldano FerroneEva A. TurleyJames B. McCarthy

cancer genomicstumor biologymelanomacancer researchcancer growthepithelial-to-mesenchymal transitioncancer biologydermatologycell biologycancer cellsdata sciencecell motilityproteomicsgene expressionmedicinal researchtumor cell motility

Data From Nfat1 Directly Regulates Il8 and Mmp3 to Promote Melanoma Tumor Growth and Metastasis. (2023)

metastasistumor biologycancer growthcancer biologycell biologycell signalingcancer cellsoncogenesmelanomapathologymelanoma tumor growth

Data From Overexpression of Protease-Activated Receptor-1 Contributes to Melanoma Metastasis via Regulation of Connexin 43. (2023)

metastasistumor biologyconnexin 43cancer biologyprotease-activated receptor-1 contributescell biologycell signalingcancer cellsoncogenesimmunologymelanoma metastasissignal transductionmelanoma

Data From Therapeutic Destruction of Insulin Receptor Substrates for Cancer Treatment. (2023)

insulin managementinsulin signalingcancer researchcell biologydrug targetsbiochemistrypharmacologyinsulin receptor substratestherapeutic targetscancer treatmenttherapeutic destructiondiabetes therapiesdiabetes research

Supplementary Figure 1 From Uncoupling Between Epidermal Growth Factor Receptor and Downstream Signals Defines Resistance to the Antiproliferative Effect of Gefitinib in Bladder Cancer Cells. (2023)

Wassim KassoufColin P.N. DinneyGordon BrownDavid J. McConkeyAlan J. DiehlMenashe Bar-EliLiana Adam

bladder cancer cellsurologyreceptorsdownstream signalscancer researchcancer biologydrug resistancebladder cancerpharmacologyantiproliferative effect

Supplementary Figure 2 From Nfat1 Directly Regulates Il8 and Mmp3 to Promote Melanoma Tumor Growth and Metastasis. (2023)

metastasistumor biologysignaling pathwayscancer growthcancer biologycell biologycell signalingcancer cellsoncogenesmelanomasupplementary figure 2pathologymelanoma tumor growth

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Menashe Bar-Eli

The University of Texas MD Anderson Cancer Center

Affiliations

Center For Rna Interference And Non-Coding Rna

Department Of Cancer Biology

MD Anderson UTHealth Houston Graduate School

The University of Texas MD Anderson Cancer Center

NoneMenashe Bar-eli is currently serving as a Center For Rna Interference And Non-Coding Rna faculty at The University of Texas Md Anderson Cancer Center.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Menashe Bar-Eli

The University of Texas MD Anderson Cancer Center

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Abstract 5073: Nt219 Induces Tumor Pd-L1 Expression and Potentiates Anti-Pd-1 Efficacy. Cancer Research (2023)

Ricardo Alexandre de AzevedoHadas ReuveniMenashe Bar-EliMichael Curran

tumor biologygene expressiononcologycancer researchcancer biologymolecular biologytumor microenvironment

Conflict of Interest Form 1 From Mir-200 Expression Regulates Epithelial-To-Mesenchymal Transition in Bladder Cancer Cells and Reverses Resistance to Epidermal Growth Factor Receptor Therapy. (2023)

bladder cancer cellsurologytumor biologymolecular cancercancer biologycell biologycancer cellsmolecular cell biologyoncogenesreverses resistancebladder cancersmall rnainterest form 1

Data From a Subset of Host B Lymphocytes Controls Melanoma Metastasis Through a Melanoma Cell Adhesion Molecule/Muc18-Dependent Interaction: Evidence From Mice and Humans. (2023)

metastasisimmunomodulationtumor biologyimmune responsescell biologycancer cellsa subsetcancer immunosurveillanceimmunologycell researchmelanomatumor microenvironment

Data From Expression of Id-1 is Regulated by Mcam/Muc18: A Missing Link in Melanoma Progression. (2023)

Maya ZiglerGabriel J. VillaresAndrey S. DobroffHua WangLi HuangRussell R. BraeuerTakafumi KamiyaVladislava O. MelnikovaRenduo SongRan FriedmanRhoda M. AlaniMenashe Bar-Eli

tumor biologygeneticsmelanoma progressionmelanomacancer researchoncologycancer biologydermatologydata sciencegene expressionskin cancercancer genetics

Data From Melanoma Proteoglycan Modifies Gene Expression to Stimulate Tumor Cell Motility, Growth, and Epithelial-To-Mesenchymal Transition. (2023)

Jianbo YangMatthew A. PriceGui Yuan LiMenashe Bar-EliRavi SalgiaRamasamy JagedeeswaranJennifer H. CarlsonSoldano FerroneEva A. TurleyJames B. McCarthy

Data From Nfat1 Directly Regulates Il8 and Mmp3 to Promote Melanoma Tumor Growth and Metastasis. (2023)

metastasistumor biologycancer growthcancer biologycell biologycell signalingcancer cellsoncogenesmelanomapathologymelanoma tumor growth

Data From Overexpression of Protease-Activated Receptor-1 Contributes to Melanoma Metastasis via Regulation of Connexin 43. (2023)

metastasistumor biologyconnexin 43cancer biologyprotease-activated receptor-1 contributescell biologycell signalingcancer cellsoncogenesimmunologymelanoma metastasissignal transductionmelanoma

Data From Therapeutic Destruction of Insulin Receptor Substrates for Cancer Treatment. (2023)

Supplementary Figure 1 From Uncoupling Between Epidermal Growth Factor Receptor and Downstream Signals Defines Resistance to the Antiproliferative Effect of Gefitinib in Bladder Cancer Cells. (2023)

Wassim KassoufColin P.N. DinneyGordon BrownDavid J. McConkeyAlan J. DiehlMenashe Bar-EliLiana Adam

bladder cancer cellsurologyreceptorsdownstream signalscancer researchcancer biologydrug resistancebladder cancerpharmacologyantiproliferative effect

Supplementary Figure 2 From Nfat1 Directly Regulates Il8 and Mmp3 to Promote Melanoma Tumor Growth and Metastasis. (2023)

metastasistumor biologysignaling pathwayscancer growthcancer biologycell biologycell signalingcancer cellsoncogenesmelanomasupplementary figure 2pathologymelanoma tumor growth

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5
19
10 / page