Faculty

Institution

Unit

Venue

Mahsa Sadeghzadeh

Upstate Medical University

Affiliations

Norton College of Medicine

Department Of Medicine

Division Of Rheumatology

Department Of Rheumatology

Department Of Medicine

Division Of Rheumatology

NoneMahsa Sadeghzadeh is currently serving as a Medicine faculty at Upstate Medical University.

Honors

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Experiences

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Education

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Mahsa Sadeghzadeh

Upstate Medical University

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immunology cell biology immune responses inflammation cell metabolism cellular immune responses systems biology epidemiology gene expression pathogens antimicrobial resistance microbial pathogenesis drug resistance pathogenic bacteria autophagy antimicrobial agents infection control clinical microbiology applied microbiology hospital infections cell death experimental medicine cell research molecular cell biology mitochondrial dynamics mitochondrial function mitophagy emerging infectious diseases infectious respiratory disease disinfectants antibacterial agents antimicrobial chemotherapy cell regulation adaptive immune system kynurenine-sensitive mtor activation pro-inflammatory mitochondrial metabolism cd38-dependent nad+ metabolism

Notable Scholarly Works

Notable Federal Grants

Collaborators

Rab4A Drives Proinflammatory Cd4+ T Cell Signaling via Cd38-Dependent Nad+ Metabolism. Research Square (2025)

immunologycell deathcell regulationcellular immune responsesexperimental medicinecell signalingcell researchmolecular cell biologyadaptive immune systemcd38-dependent nad+ metabolismmolecular biologyimmune responsescell biologysignal transductioncell metabolismmetabolic pathwaysinflammation

Eradication of Pseudomonas Aeruginosa Persister Cells by Eravacycline. ACS Infectious Diseases (2024)

Sweta RoyZeynep S. CakmakSalma MahmoudMahsa SadeghzadehGuirong WangDacheng Ren

pathogensantimicrobial agentsantimicrobial resistancemicrobial pathogenesisemerging infectious diseasesinfection controlepidemiologyclinical microbiologyapplied microbiologyinfectious respiratory diseasedisinfectantsdrug resistancepathogenic bacteriahospital infectionsantibacterial agentsinfectious diseasesmicrobiologyantimicrobial chemotherapy

Rab4A-Directed Endosome Traffic Shapes Pro-Inflammatory Mitochondrial Metabolism in T Cells via Mitophagy, Cd98 Expression, and Kynurenine-Sensitive Mtor Activation. Nature Communications (2024)

Nick HuangThomas WinansBrandon WymanZachary OaksTamas FaludiGourav ChoudharyZhi-Wei LaiJoshua LewisMiguel BeckfordManuel DuarteDaniel KrakkoAkshay PatelJoy ParkTiffany CazaMahsa SadeghzadehLaurence MorelMark HaasFrank MiddletonKatalin BankiAndras Perl

immunologygene expressionmitochondrial dynamicskynurenine-sensitive mtor activationcellular immune responsessystems biologymitochondrial functionpro-inflammatory mitochondrial metabolismimmune responsesmitophagycell biologycell metabolismautophagyinflammation

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Mahsa Sadeghzadeh

Upstate Medical University

Affiliations

Norton College of Medicine

Department Of Medicine

Division Of Rheumatology

Department Of Rheumatology

Department Of Medicine

Division Of Rheumatology

NoneMahsa Sadeghzadeh is currently serving as a Medicine faculty at Upstate Medical University.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Mahsa Sadeghzadeh

Upstate Medical University

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Rab4A Drives Proinflammatory Cd4+ T Cell Signaling via Cd38-Dependent Nad+ Metabolism. Research Square (2025)

Eradication of Pseudomonas Aeruginosa Persister Cells by Eravacycline. ACS Infectious Diseases (2024)

Sweta RoyZeynep S. CakmakSalma MahmoudMahsa SadeghzadehGuirong WangDacheng Ren

Rab4A-Directed Endosome Traffic Shapes Pro-Inflammatory Mitochondrial Metabolism in T Cells via Mitophagy, Cd98 Expression, and Kynurenine-Sensitive Mtor Activation. Nature Communications (2024)

1
10 / page