Faculty

Institution

Unit

Venue

Ma, Rong

University of North Texas Health Science Center

Affiliations

College of Biomedical and Translational Sciences

Microbiology, Immunology and Genetics

Physiology and Anatomy

University of North Texas Health Science Center

College of Health Professions

Texas Prevention Institute (TPI)

School Of Public Health

Department Of Behavioral And Community Health

Department Of Health Administration And Health Policy

Department Of Health Behavior And Health Systems

Department Of Population And Community Health

Texas College of Osteopathic Medicine

Department of Internal Medicine

UNT System College of Pharmacy

Pharmacotherapy

NoneMa, Rong is currently serving as a College Of Biomedical And Translational Sciences faculty at University of North Texas Health Science Center.

Honors

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Experiences

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Education

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Ma, Rong

University of North Texas Health Science Center

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cell biology physiology cell signaling signal transduction cellular physiology molecular physiology nephrology renal pathophysiology cell physiology vascular biology pharmacology renal function diabetes glomerular mesangial cells receptors molecular biology diabetes research molecular and cellular biology kidney research mesangial cells electrophysiology membrane biology molecular cell biology cell metabolism insulin signaling cell death diabetic medicine redox signaling free radical biology diabetes therapies redox biology receptor (biochemistry) signaling pathways store-operated calcium entry cellular biochemistry reactive oxygen species circulatory physiology liver physiology g protein-coupled receptor intracellular trafficking cell regulation diabetic kidney disease high glucose biological chemistry rna biology cell growth cellular biology cell interactions apoptosis cell trafficking metabolic regulation pharmacological research vascular pharmacology cell-matrix interactions cytokines molecular communication molecular regulation ion channels epidermal growth factor angiotensin ii acute effects biomolecular analysis cancer biology long non-coding rna functional selectivity physiological research therapeutic nanomaterials pharmaceutical nanotechnology lncrna-mirna-mrna network molecular determinants vascular myocytes redox-regulated cation channel polycationic cyclodextrin nanoparticles sirna delivery glomerular mesangium high glucose conditions

Notable Scholarly Works

Notable Federal Grants

Collaborators

I-Mfa, Mesangial Cell Trpc1 Channel, and Regulation of Gfr. Journal of the American Society of Nephrology (2024)

cell physiologymolecular and cellular biologycell signalingcell biologytranscription factorscellular physiologymolecular physiologymolecular cell biologymolecular pathwayssignal transductionreceptors

Non-Voltage-Gated Ca 2+ Channel Signaling in Glomerular Cells in Kidney Health and Disease. American Journal of Physiology-Renal Physiology (2024)

Rong MaYu TaoMichael L. WadeRobert T. Mallet

kidney healthnephrologyphysiologycell signalingelectrophysiologyneurosciencesignaling pathwayssignal transductionglomerular cellsurology

Reconstituted Hdl Ameliorated Renal Injury of Diabetic Kidney Disease in Mice. Physiological Reports (2024)

vascular biologydiabetic kidney diseasenephrologyrenal injurytranslational medicinediabetes researchpharmacologyrenal pathophysiologyurologydiabeteschronic kidney diseasediabetes therapies

Sex and Strain Differences in Renal Hemodynamics in Mice. Physiological Reports (2023)

reproductive biologyrenal functionnephrologyphysiologyhuman biologyrenal pathophysiologyurologystrain differences

Store-Operated Calcium Entry Inhibition Ameliorates Ang II Induced Podocyte Apoptosis and Mitochondria Respiratory Dysfunction. Physiology (2023)

cell physiologyinflammationapoptosisang iiphysiologycell deathcell signalingpodocyte apoptosiscell biologycellular physiologymolecular physiologymitochondria respiratory dysfunctionpharmacologyoxidative stresssignal transduction

Store-Operated Ca 2+ Entry Inhibition Ameliorates High Glucose and Ang II-Induced Podocyte Apoptosis and Mitochondrial Damage. American Journal of Physiology-Renal Physiology (2023)

cell physiologyapoptosisphysiologycell deathcell signalinghigh glucosecell biologycellular physiologymitochondrial damagesignal transduction

Enhanced Orai1-Mediated Store-Operated Ca2+ Channel/Calpain Signaling Contributes to High Glucose-Induced Podocyte Injury. Journal of Biological Chemistry (2022)

cell physiologymolecular and cellular biologyphysiologycell signalingcell biologycellular physiologymolecular physiologypharmacologyinsulin signalingsignal transductioncellular biochemistry

Exaggerated Store‐Operated Calcium Entry: A Mechanism for High Glucose‐Induced Podocyte Injury and Mitochondria Damage. The FASEB Journal (2022)

cell physiologymetabolismosteoporosisphysiologycellular physiologybiochemistryelectrolyte disturbancestore‐operated calcium entrymitochondria damage

Ion Channels and Channelopathies in Glomeruli. Physiological Reviews (2022)

Alexander StaruschenkoRong MaOleg PalyginStuart E. Dryer

neurologyphysiological researchphysiologyelectrophysiologyneuroscienceneurophysiologyion channels

Neogenin Pathway Positively Regulates Fibronectin Production by Glomerular Mesangial Cells. American Journal of Physiology-Cell Physiology (2022)

vascular biologyrenal functionnephrologycell-matrix interactionscell signalingcell biologycellular physiologydevelopmental biologymolecular physiologyrenal pathophysiologyneogenin pathwayextracellular matrixcellular biochemistry

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Ma, Rong

University of North Texas Health Science Center

Affiliations

College of Biomedical and Translational Sciences

Microbiology, Immunology and Genetics

Physiology and Anatomy

University of North Texas Health Science Center

College of Health Professions

Texas Prevention Institute (TPI)

School Of Public Health

Department Of Behavioral And Community Health

Department Of Health Administration And Health Policy

Department Of Health Behavior And Health Systems

Department Of Population And Community Health

Texas College of Osteopathic Medicine

Department of Internal Medicine

UNT System College of Pharmacy

Pharmacotherapy

NoneMa, Rong is currently serving as a College Of Biomedical And Translational Sciences faculty at University of North Texas Health Science Center.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Ma, Rong

University of North Texas Health Science Center

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

I-Mfa, Mesangial Cell Trpc1 Channel, and Regulation of Gfr. Journal of the American Society of Nephrology (2024)

cell physiologymolecular and cellular biologycell signalingcell biologytranscription factorscellular physiologymolecular physiologymolecular cell biologymolecular pathwayssignal transductionreceptors

Non-Voltage-Gated Ca 2+ Channel Signaling in Glomerular Cells in Kidney Health and Disease. American Journal of Physiology-Renal Physiology (2024)

Rong MaYu TaoMichael L. WadeRobert T. Mallet

kidney healthnephrologyphysiologycell signalingelectrophysiologyneurosciencesignaling pathwayssignal transductionglomerular cellsurology

Reconstituted Hdl Ameliorated Renal Injury of Diabetic Kidney Disease in Mice. Physiological Reports (2024)

vascular biologydiabetic kidney diseasenephrologyrenal injurytranslational medicinediabetes researchpharmacologyrenal pathophysiologyurologydiabeteschronic kidney diseasediabetes therapies

Sex and Strain Differences in Renal Hemodynamics in Mice. Physiological Reports (2023)

reproductive biologyrenal functionnephrologyphysiologyhuman biologyrenal pathophysiologyurologystrain differences

Store-Operated Calcium Entry Inhibition Ameliorates Ang II Induced Podocyte Apoptosis and Mitochondria Respiratory Dysfunction. Physiology (2023)

Store-Operated Ca 2+ Entry Inhibition Ameliorates High Glucose and Ang II-Induced Podocyte Apoptosis and Mitochondrial Damage. American Journal of Physiology-Renal Physiology (2023)

cell physiologyapoptosisphysiologycell deathcell signalinghigh glucosecell biologycellular physiologymitochondrial damagesignal transduction

Enhanced Orai1-Mediated Store-Operated Ca2+ Channel/Calpain Signaling Contributes to High Glucose-Induced Podocyte Injury. Journal of Biological Chemistry (2022)

cell physiologymolecular and cellular biologyphysiologycell signalingcell biologycellular physiologymolecular physiologypharmacologyinsulin signalingsignal transductioncellular biochemistry

Exaggerated Store‐Operated Calcium Entry: A Mechanism for High Glucose‐Induced Podocyte Injury and Mitochondria Damage. The FASEB Journal (2022)

cell physiologymetabolismosteoporosisphysiologycellular physiologybiochemistryelectrolyte disturbancestore‐operated calcium entrymitochondria damage

Ion Channels and Channelopathies in Glomeruli. Physiological Reviews (2022)

Alexander StaruschenkoRong MaOleg PalyginStuart E. Dryer

neurologyphysiological researchphysiologyelectrophysiologyneuroscienceneurophysiologyion channels

Neogenin Pathway Positively Regulates Fibronectin Production by Glomerular Mesangial Cells. American Journal of Physiology-Cell Physiology (2022)

1
2
3
4
5
9
10 / page