Faculty

Institution

Unit

Venue

Gary ZeRuth

Murray State University

Affiliations

Department Of Biology

Murray State University

NoneGary Zeruth is currently serving as a Biology faculty at Murray State University.

Honors

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Experiences

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Education

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Gary ZeRuth

Murray State University

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cell biology gene expression molecular biology transcription factors molecular cell biology molecular and cellular biology cell signaling molecular regulation developmental biology signal transduction transcription (biology) cell metabolism diabetes research gene regulation biochemistry molecular genetics diabetes molecular physiology cellular biochemistry developmental system developmental cell protein function gene function insulin signaling cell fate determination insulin gene expression structural molecular biology molecular metabolism pediatric diabetes nucleic acids oncogenes biological chemistry thyroid cellular physiology genome biology protein expression metabolic regulation diabetic kidney disease molecular basis gene regulatory networks transcription regulation thyroid hormone genetic disorders embryonic development signaling pathways cell regulation diabetes mellitus crispr gene editing genetic technologies pancreatic b-cell development novel critical player insulin transcription key regulator physiological functions cystic renal disease neonatal diabetes transactivating mechanisms dna binding nuclear localization novel negative regulator transcriptional activity gli-similar 3 transactivation function zebrafish embryos

Notable Scholarly Works

Notable Federal Grants

Collaborators

Downregulation of Glis3 in Ins1 Cells Exposed to Chronically Elevated Glucose Contributes to Glucotoxicity-Associated β Cell Dysfunction. Islets (2024)

glucose contributesins1 cellsdiabetes mellitusphysiologycell signalingcell biologycell metabolismdiabetes researchinsulin signalingdiabetes

Whole Mount Immunohistochemistry in Zebrafish Embryos and Larvae. Journal of Visualized Experiments (2020)

embryologyembryonic developmentdevelopmental cellcell biologyzebrafish embryosdevelopmental biologycell fate determinationdevelopmental systemtissue (biology)

Pias-Family Proteins Negatively Regulate Glis3 Transactivation Function Through Sumo Modification in Pancreatic β Cells. Heliyon (2018)

sumo modificationglis3 transactivation functioncell signalingcell biologycellular physiologysignaling pathwayscell metabolismmolecular cell biologygene functionmolecular biologypias-family proteinssignal transductiongene expressioncell regulationcellular biochemistry

Generating a Transgenic Zebrafish Line Using Crispr/Cas9 Genome Editing to Determine the Spatial and Temporal Expression of Transcription Factor Glis3 During Development. Murray State's Digital Commons (Murray State University) (2017)

gene editinggenetic engineeringgenome editingtranscription factor glis3developmental cellgenetic technologiescrisprcell biologytranscription factorsgenome biologycrispr/cas9 genomedevelopmental biologymolecular cell biologycell fate determinationdevelopmental systemgene expressiontransgenic zebrafish linegenetics

Hect E3 Ubiquitin Ligase Itch Functions as a Novel Negative Regulator of Gli-Similar 3 (Glis3) Transcriptional Activity. PLoS ONE (2015)

transcriptional activitymolecular and cellular biologyoncogenescell signalinggli-similar 3cell biologytranscription factorsnovel negative regulatorgene regulationmolecular cell biologygene functiontranscription (biology)molecular biologytranscription regulationmolecular regulationgene expressiongene regulatory networks

Transcription Factor Gli-Similar 3 (Glis3): Implications for the Development of Congenital Hypothyroidism.. PubMed (2014)

genetic disorderscellular differentiationthyroid hormonetranscription factorsdevelopmental biologyhuman geneticsendocrinologygene expressionthyroidgenetics

The KrüPpel-Like Protein Gli-Similar 3 (Glis3) Functions as a Key Regulator of Insulin Transcription. Molecular Endocrinology (2013)

molecular metabolismcell signalingmetabolic regulationcell biologytranscription factorsdiabetes researchcell metabolismmolecular physiologymolecular cell biologygene regulationtranscription (biology)molecular biologyinsulin signalinginsulin transcriptionsignal transductiongene expressionmolecular regulationkey regulator

Gli-Similar Proteins. Vitamins and hormones (2012)

Modulation of the Transactivation Function and Stability of KrüPpel-Like Zinc Finger Protein Gli-Similar 3 (Glis3) by Suppressor of Fused. Journal of Biological Chemistry (2011)

molecular and cellular biologycell signalingcell biologytranscription factorsprotein functiontransactivation functionmolecular cell biologymolecular biologybiological chemistrysignal transductiongene expressionmolecular regulation

molecular and cellular biologydiabetic kidney diseaseproteomicscell signalingmolecular medicinecell biologypediatric diabetesbiochemistrydiabetes researchmolecular physiologycystic renal diseasemolecular biologyneonatal diabetessignal transductionphysiological functionsdiabetes

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Gary ZeRuth

Murray State University

Affiliations

Department Of Biology

Murray State University

NoneGary Zeruth is currently serving as a Biology faculty at Murray State University.

Honors

Create free account to unlock more features

Experiences

Create free account to unlock more features

Education

Create free account to unlock more features

Gary ZeRuth

Murray State University

You can click a keyword to filter the displayed items

Notable Scholarly Works

Notable Federal Grants

Collaborators

Downregulation of Glis3 in Ins1 Cells Exposed to Chronically Elevated Glucose Contributes to Glucotoxicity-Associated β Cell Dysfunction. Islets (2024)

glucose contributesins1 cellsdiabetes mellitusphysiologycell signalingcell biologycell metabolismdiabetes researchinsulin signalingdiabetes

Whole Mount Immunohistochemistry in Zebrafish Embryos and Larvae. Journal of Visualized Experiments (2020)

embryologyembryonic developmentdevelopmental cellcell biologyzebrafish embryosdevelopmental biologycell fate determinationdevelopmental systemtissue (biology)

Pias-Family Proteins Negatively Regulate Glis3 Transactivation Function Through Sumo Modification in Pancreatic β Cells. Heliyon (2018)

Hect E3 Ubiquitin Ligase Itch Functions as a Novel Negative Regulator of Gli-Similar 3 (Glis3) Transcriptional Activity. PLoS ONE (2015)

Transcription Factor Gli-Similar 3 (Glis3): Implications for the Development of Congenital Hypothyroidism.. PubMed (2014)

genetic disorderscellular differentiationthyroid hormonetranscription factorsdevelopmental biologyhuman geneticsendocrinologygene expressionthyroidgenetics

The KrüPpel-Like Protein Gli-Similar 3 (Glis3) Functions as a Key Regulator of Insulin Transcription. Molecular Endocrinology (2013)

Gli-Similar Proteins. Vitamins and hormones (2012)

Modulation of the Transactivation Function and Stability of KrüPpel-Like Zinc Finger Protein Gli-Similar 3 (Glis3) by Suppressor of Fused. Journal of Biological Chemistry (2011)

1
2
10 / page