Abe Clark

Executive Director and FARVOProfessor North Texas Eye Research Institute ; Executive Director and Regents Professor Pharmacology & Neuroscience

University of North Texas Health Science Center

Affiliations

College of Biomedical and Translational Sciences

Pharmacology and Neuroscience

Division of Research and Innovation

North Texas Eye Research Institute

Graduate School of Biomedical Sciences ; North Texas Eye Research Institute ; Pharmacology & Neuroscience

University of North Texas Health Science Center

North Texas Eye Research Institute (NTERI)

School of Biomedical Sciences

Pharmacology and Neuroscience

Texas College of Osteopathic Medicine

UNT System College of Pharmacy

Abe Clark is currently serving as a College Of Biomedical And Translational Sciences Executive Director and FARVOProfessor North Texas Eye Research Institute ; Executive Director and Regents Professor Pharmacology & Neuroscience at University of North Texas Health Science Center.

Honors

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Experiences

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Education

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Abe Clark

University of North Texas Health Science Center

Executive Director and FARVOProfessor North Texas Eye Research Institute ; Executive Director and Regents Professor Pharmacology & Neuroscience

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ophthalmology ophthalmology retina glaucoma ocular surface cellular physiology trabecular meshwork ocular disease neuro-ophthalmology cell research experimental ophthalmology ocular surface physiology ocular tissues retina signal transduction molecular physiology optic nerve ocular pathology pharmacology ganglion cell neuroscience neurons gene expression glucocorticoids molecular neurodegeneration aqueous humour intraocular pressure molecular diagnostics systems biology transcription (biology) ciliary body transcriptome pharmacological research genome biology cell physiology signaling pathways lamina cribrosa cells medicinal research animal biotechnology neuroregeneration aldehyde dehydrogenase neovascularization neuropharmacology hyperpolarization (biology) mechanotransduction mammalian physiology neuropsychopharmacology photomedicine applied pharmacology biomedical analysis heredity neuroprotection gene expression profiling forensic identification cell detection genotype-phenotype association genome-wide association studies disease gene identification corneal dystrophy alcohol dehydrogenases cell manipulation embryonic development angiogenesis novel neuroprotective compound rabbit animal models abnormal calcium homeostasis glucocorticoid responder gene expression profiles membrane stress stretch-activated channels rat intraocular pressure adenoviral gene transfer rodent eyes glaucoma therapeutics forensic path age-related permeability changes 3-hydroxysteroid dehydrogenases cell line glucocorticoid receptors nerve growth factors retinal ganglion cells eye diseases western blotting aqueous humor extracellular matrix proteins protein isoforms optic disk optic nerve diseases messenger rna bone morphogenetic proteins ocular hypertension extracellular matrix

Notable Scholarly Works

Notable Federal Grants

Collaborators

Delivery of a Novel Neuroprotective Compound to the Retina in Rat and Rabbit Animal Models. Journal of Controlled Release (2025)

rabbit animal modelsneuropharmacologyneuronspharmacological researchneuroprotectionneuro-ophthalmologyneuroscienceophthalmology retinaretinapharmacologyophthalmologymolecular neurodegenerationphotomedicineexperimental ophthalmologynovel neuroprotective compoundneuropsychopharmacology

Glucocorticoid Receptor Transactivation is Required for Glucocorticoid-Induced Ocular Hypertension and Glaucoma. Investigative Ophthalmology & Visual Science (2019)

receptorsocular diseaseocular tissuesendocrinologyglucocorticoid receptor transactivationglaucomaophthalmology retinaocular surface physiologyglucocorticoid-induced ocular hypertensionphysiologyophthalmologyglucocorticoidsocular surface

Letter to the Editor Concerning Webber Et Al. Exp Eye Res. 148: 97-102, 2016. (2019)

visual systemocular diseasevisual sciencedermatologyneuro-ophthalmologyocular pathologyophthalmology retinaeye trackingophthalmologyvision rehabilitationexperimental ophthalmologyocular surfaceexp eye reseye health

Activation of the Nfat-Calcium Signaling Pathway in Human Lamina Cribrosa Cells in Glaucoma. Investigative Ophthalmology and Visual Science (2018)

signaling pathwayscell physiologycell biologycell signalingglaucomaphysiologysignal transductionciliary bodycellular physiologymolecular physiology

Bmp and Activin Membrane Bound Inhibitor Regulates the Extracellular Matrix in the Trabecular Meshwork. Investigative Ophthalmology and Visual Science (2018)

developmental biologytrabecular meshworkcell biologycell signalingmolecular cell biologydevelopmental cellcell-matrix interactionsextracellular matrixmatrix biologysignal transductioncellular physiology

Consensus Recommendations for Trabecular Meshwork Cell Isolation, Characterization and Culture.. (2018)

cytoskeletontrabecular meshworkcell cultureconsensus recommendationscell biologytissue culturebiomedical engineeringcell researchtissue (biology)ocular surface

Establishment of a Conditionally Immortalized Mouse Optic Nerve Astrocyte Line. Experimental eye research (2018)

neuropathologymolecular neuroscienceneuronsoptic nerveneuro-ophthalmologyneuroscienceophthalmology retinaretinabrain tumor researchophthalmologyganglion cell

Glucocorticoid Receptor Gr Regulates Glucocorticoid-Induced Ocular Hypertension in Mice. Scientific Reports (2018)

receptorsocular diseaseglucocorticoid receptor grendocrinologyneuro-ophthalmologyhypertensionophthalmology retinavascular biologypsychoneuroendocrinologyphysiologyophthalmologyendocrine hypertensionglucocorticoidsocular surface

Histological Investigation of Human Glaucomatous Eyes: Extracellular Fibrotic Changes and Galectin 3 Expression in the Trabecular Meshwork and Optic Nerve Head. Clinical Anatomy (2018)

optic nerve headtrabecular meshworkocular tissuesgalectin 3dermatologyocular pathologyglaucomaophthalmology retinahuman glaucomatous eyestissue (biology)ophthalmologyocular surface

Knockout of Tissue Transglutaminase Ameliorates Tgf2-Induced Ocular Hypertension: A Novel Therapeutic Target for Glaucoma?. Experimental Eye Research (2018)

ocular diseasetgf2-induced ocular hypertensionocular pathologyglaucomaophthalmology retinapharmacologynovel therapeutic targettherapeutic targetsophthalmologyocular surfacetissue transglutaminase

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